The Patient Power Association

Please note that Damien passed away on 10.9.2025. His wife Ivana will be updating this website and keeping this blog active as soon as she is able to and is not overcome with grief, after 25 wonderful years spent with Damien. Ivana is also writing a book on techniques Damien used to treat scoliosis and back pain as well as a book about supplements and therapies mentioned below in this particular blog post. 

 

My Name is Damien Mearns, CEO of the Patient Power Association (PPA).

Welcome.                                                                                                               

In the world of medicine there are power blocks: The Big Pharma companies, Medical Equipment companies, The Doctors, the Hospitals, the Government and the Patient.

In the reality of things, the whole set up is meant to be for the benefit of the patient. But the patient has the weakest voice at the table and rarely, if ever, even has a place at the table.

The position of the patient is the position of a medieval serf supplicating his master, begging him from crumbs out of pity.

This is 2025… this is not good enough. You might though ask: Why is this important ? Aren’t they all doing their best anyway ? wouldn’t we just get in the way ?

No. Last week, Mid-March 2025, I found out I had stomach cancer. Up to that point I was a back therapist using the ASMI back fixing machine so I can show you visibly here the difference: On the left is a result of “letting them do their best” and on the right is “us getting involved”:

I had scoliosis, diagnosed in teenage years, I got treated with the ASMI – the results were brilliant – life changing in a few sessions. No hospital on the planet will be able to get the results you see here using their current techniques – Henry’s Before and After’s shown above are just one of many improvements you can see on this website. (GoodBack.co.uk)

I have been trying for 6 years to get these techniques into the hospitals, the doctors… everywhere. I show the techniques for free on the website for the world to copy.

From the “Patient Power” perspective, the need is to get this patient’s back better as quick as possible and as safely as possible – these are the patient’s “interests”.

For the other players, the patient on the left is an income source, an experimental subject, a voter… As Patient Power does not have a voice at the table, no hospital will ever have any interest in treating their patients with these simple (no back cracking, nor surgery, no pills…) techniques as it does not align with the interests of any of the big players.

The epistemological vanity of the medical world.

Epistemology is the study of how to we know things, it was an ancient Greek problem they never solved back then. When you chat to doctors, they use phrases like: “We Know that so and so happens… “. When they do so, especially when they punch the word “Know”, they mean – full sentence: “This has been proved via a large multi-centred multi-$Million randomised double blind controlled trial” – this is also known as a Phase 3 trial. Everything else, other than this, is of little to no worth in terms of being called “evidence”, in terms of “Knowing” . As far as doctors are concerned they’ve got Epistemology licked: If you want to Know something: do a phrase 3 trial on it.

This is plain wrong – so says D L Sackett the “Father of Evidence based medicine”: 

“The practice of evidence-based medicine means integrating individual clinical expertise with the best available external clinical evidence from systematic research. By individual clinical expertise we mean the proficiency and judgment that we individual clinicians acquire through clinical experience and clinical practice. Increased expertise is reflected in many ways, but especially in more effective and efficient diagnosis and in the more thoughtful identification and compassionate use of individual patients’ predicaments, rights, and preferences in making clinical decisions about their care. By best available external clinical evidence we mean clinically relevant research, often from the basic sciences of medicine, but especially from patient centered clinical research into the accuracy and precision of diagnostic tests (including the clinical examination), the power of prognostic markers, and the efficacy and safety of therapeutic, rehabilitative, and preventive regimens. External clinical evidence both invalidates previously accepted diagnostic tests and treatment and replaces them with new ones that are more powerful, more accurate, more efficacious, and safer. Good doctors use both individual clinical expertise and the best available external evidence, and neither alone is enough. Without clinical expertise, practice risks becoming tyrannized by external evidence, for even excellent external evidence may be inapplicable to or inappropriate for an individual patient.”

Big Pharma have redefined Evidence based medicine and turned it into the very tyranny that Sackett warned about. This tyranny is run, like much of the modern world, from a lawyers office somewhere on Wall Street. National and International regulators for all treatments round the globe are largely funded and hence controlled by this same central interest group:

Their replies to this are 1) The headline that appears on a Google search of this issue which is: “Oh no we don’t”… “The MHRA does not receive financial contributions or donations” and 2) What they say when you actually click that headline and read the article: “OK, actually we do  we do” … “We do receive funding from the Bill and Melinda Gates Foundation as well as other sources outside government such as WHO. This funding mainly supports work to strengthen regulatory systems in other countries. The majority of our income comes from the pharmaceutical industry through fees”

Professor Robert Llewellyn Clancy AM is a leading Australian clinical immunologist and a pioneer in the field of mucosal immunology (260 Publications): 

“The Random Control Trial (RCT) has become the tool of the pharmaceutical industry as expensive, targeted studies geared to selectively support their drug of the day.  Covid became a field day for dodgy RCTs – from hidden and blurred  data in the initial mRNA vaccine trials, withdrawal of published RCT pertaining to show hydroxychloroquine (HCQ) did not work on the basis of falsified data, the plethora of documented faults in those studies that remain such as the “Together Trial” labelled as “fraudulent” for data manipulation and over 50 significant defects including “loss” of half the placebo group; and the recent withdrawal of a Cochrane Analysis for ivermectin due to bias and selective handling of data (and following numerous complaints). The argument that only RCT’s can be used to assess clinical validity is not only specious and convenient to criticise repurposed drugs which will never have the financial support of the pharmaceutical industry, but patently incorrect.  I shared medical intake at MacMaster University with Dave Sackett for 5 years.  Dave, the “Father of EBM” would lecture me nearly daily on evidence assessment.  His point was always that evidence-based decisions were based on three platforms: the best published evidence in its entirety – (not just Phase 3 RCT’s) ; input from experienced clinicians; and patient expectations.” – 

So the claim that: “It’s simple: A Phase 3 trial = what we Know, nothing else gets close. End of discussion” is propaganda-level nonsense. Let’s take a metaphor – Seatbelts: Someone (it was Volvo) invented the seat belt. For me this is the clever bit, the science & engineering – this is the “clinical trial” stage. They gave it to the world. The next question is “will the regulators allow these seatbelts to be used” ? 

In the real world, the regulators took a look at it and said “Yes”, this saved many lives over the years.

What would have happened if the regulators had been trained in the medical world ? They would say: “Well we can see it works from the “Clinical trials” , but we don’t Know it works and we won’t authorise it’s use until we have this scientific proof, this “know” i.e. a “phase 3 trial”. So until a large “large multi-centred multi-$Billion randomised double blind controlled trial” proves they are safe & effective, “we will not authorise seatbelts”.

Cost of this seatbelt trial trial :

Phase 3 trials cost multi-$millions , $20 million would be low. So when they say “Show us your evidence” , “it’s not proven”, “Stick it in a “Peer Reviewed Scientific Journal” they mean “Show us the money… lots and lots of money”.

But that is the least of the costs: Road accidents kill large numbers each year. Many of these lives could be saved if the seatbelts were authorised immediately. Just the waiting alone for the trials to get started, which can take decades, and won’t take place at all unless the huge funding is in place. And this won’t happen at all unless that funding can return a huge profit for the funder. So while we wait for “further research” “to do it properly”, “just to be safe” “so we Know for sure”, we have the cost of mass slaughter of car users. Then, when the trial starts, you have to fit thousands of cars with these new real seat-belts along with thousands of fake seat-belts – the placebo – into others and sit back and watch the bodies pile up in the morgue from this callous slaughter. The body count – is then “the data” – this gets counted and analysed “Jeez George, that number is bigger than that number” and the results published. Then the “science is done”. Let’s say it was parachutes they were testing “Just to be safe” so we “Know” . What they would do is ban all parachutes in all aeroplanes as “they have yet been not proved safe” for decades till they got round to doing a “Phase 3 trial”. When they did get started, they would then chuck thousands of people out of aeroplanes all round the world, half with real parachutes and half with fake ones as  control. And they’d call that “doing the science”- this is wholescale mass murder. This is beyond ridiculous. But this is what is happening with the cancer treatments that we know – small “k” (no phase 3 trial) that are game changes for cancer. They are being banned “Just to be safe”… “until we “Know” for sure.

Ben Goldcare, author of “Bad Pharma”  says that Pharma Science is essentially easy: You test the drug against a placebo and see if it works. As you can see, this is not true. Phase 3 trials are known to be expensive in terms of both financial cost and human life so they are done 1) when there is doubt – which is not the case with IMM-101 and 2) Also to check the safety-benefit profile. But there are enough papers published on IMM-101 to show there is no doubt it works and that it is safe, so no phase 3 trial is needed.

What are these cancer treatments ? 

There are two groups, the ones from the UK and the ones from America. They both are brilliant and need to be joined together:

UK ones – based around Professor Angus Dalgleish of St George’s Hospital in London and his research team and collaborators around the planet:

The UK three: IMM-101, created by Immodulon Therapeutics Ltd,  Low dose Naltrexone.(LDN),  Calcifediol/Vitamin D

The IMM-101 is an experimental TB vaccine. By “experimental” I do not mean it’s brand new last week, they’ve been looking at it on clinical trials for two decades – small trials –  loads of lab work. The same sort of level a seat belt manufacturer would do to test a new design for a seatbelt. These clinical trial results have been published – the science is there and the results are nothing short of spectacular. But the big cruel phase 3 trial where they chuck 6,000 cancer patients out of an airplane with a fake parachute – in this case a fake IMM-101 shot and watch on as they die in agony has not been done. Historical medical records of former cancer patients alive and dead can be used as the baseline data to check against – and this could even give a more accurate base line that a placebo group. The Patient Power Association is against chucking 6,000 patients out of airplanes with fake parachutes just so some spreadsheet boy has a few data points.

Published IMM-101 study: Randomised, open-label, phase II study of gemcitabine with and without IMM-101 for advanced pancreatic cancer – PubMed

How IMM-101works: IMM-101 is a heat killed bacteria that is injected intradermally (which is a shallow, superficial injection into the dermis, which is located between the epidermis and the hypodermis – i.e. just under the skin to stimulate the myeloid dendritic cells to kick start an Innate immune boosting reaction to it) – a bit like the BCG jab for TB… and was originally invented from work to create a better BCG jab… one that can be reused time and time again as the BCG shot, good as it is, can only be given once – if you give it a second time it negatively effects the immune system. IMM-101 is such a large ugly (dead) bacteria, with so many things going on with it, that the immune system gives up trying to deal with it in a delicate sophisticated way – using the adaptive immune system – and instead responds with the innate immune system and effectively launches a raid of brute force type white blood cells : called gamma delta cells and Natural killer cells that just blast the target, they waste little or no time working out antigen this and surface protein that – they just blow it away. What this exercise does is it wakes up this part of the immune system and it is this part (the innate immune system ) that is by far the best at destroying cancer. They’ve been studying this for years, they know IMM-101 is safe and they know the results can be spectacular, these small trials have been published in scientific journals. Lawrence Tallon, head of the UK’s MHRA Medicines and Healthcare products Regulatory Agency, has enough published wok on IMM-101 in front of him on right now to sign this off immediately on a phase 4 basis, thus saving my life and the lives of millions of cancer patients round the world. At the moment I am facing death by Vogon level bureaucracy (Vogon ? from “The Hitchhiker’s Guide to the Galaxy”: 
Vogon’s are one of the most unpleasant races in the galaxy. Not actually evil, but just bad-tempered bureaucrats who are officious, and callous. They wouldn’t even lift a finger to save their own grandmothers from the ravenous Bug-Blatter Beast of Traal without orders signed in triplicate, sent in, sent back, lost, found, queried, subjected to public inquiry, lost again, and finally buried in soft peat for three months and recycled as firelighter).

Short video snippets: 

IMM-101 Doubles the effect of standard cancer treatments:

“Enhanced effect of checkpoint inhibitors when given after or together with IMM-101: significant responses in four advanced melanoma patients with no additional major toxicity” “The rapid and complete clinical responses seen in these patients may suggest that IMM-101 is activating a complementary pathway which is synergistic with CPI treatment.”  Journal of Translational Medicine published article: https://translational-medicine.biomedcentral.com/articles/10.1186/s12967-018-1602-8 

Complete response – aka cured, stage IV melanoma treated with IMM-101:

“Complete response observed in a 79 year-old male with stage IV melanoma treated with IMM-101 followed by pembrolizumab (subcutaneous disease). The subcutaneous lesions started to shrink within 4 days of the first infusion of pembrolizumab” https://www.researchgate.net/figure/Complete-reponse-observed-in-a-79year-old-male-with-stage-IV-melanoma-treated-with_fig2_327029656 

Complete response – aka cured, stage IV Pancreatic ductal cancer IMM-101:

IMM-101 is a total game changer for cancer treatment – off the scale territory: “Pancreatic ductal adenocarcinoma (PDAC) has an extremely poor prognosis. Median survival for metastatic patients is six to nine months and survivors beyond one year are exceptional. Pancreatic cancer is resistant to conventional chemotherapy and is often diagnosed at advanced stages. However, immunotherapy is a rapidly advancing new treatment modality, which shows promise in many solid tumor types.​ We present a patient with metastatic pancreatic cancer who underwent a synchronous resection of the primary tumour (pancreatoduodenectomy) and metastatic site (left hepatectomy) after multimodality neoadjuvant treatment with gemcitabine, nab-paclitaxel, and immunotherapy backbone with IMM-101 (an intradermally applied immunomodulator), as well as consolidation chemoradiation. Pathology of the specimens showed a complete response in both sites of the disease. The patient remains alive four years from the initial diagnosis and continues on maintenance immunotherapy. This exceptional response to initial chemo-immunotherapy was followed by a novel and off-protocol approach of low-dose capecitabine and IMM-101 as a maintenance strategy. The survival benefit and sustained performance status could set this as a new paradigm for the treatment of oligometastatic pancreatic cancer following response to systemic therapy and immunotherapy.​.” https://pubmed.ncbi.nlm.nih.gov/26870619/ 

” Nine-year survival in patient with aggressive disease and multiple bone metastases on presentation. Treated with IMM-101 and LDN” https://www.spandidos-publications.com/10.3892/ol.2022.13367 

“Five years stable disease with IMM-101 and LDN, without hormone treatment” https://www.spandidos-publications.com/10.3892/ol.2022.13367

Published Study: “An important feature of the mode of action of IMM-101 is its ability to activate and mature immature dendritic cells into a sub-class of dendritic cells that elicit Type-1 immune responses.”  https://icvi.org.uk/research/a-phase-ii-study-to-evaluate-the-safety-and-efficacy-of-imm-101-in-combination-with-checkpoint-inhibitors-in-patients-with-advanced-melanoma-final-results-of-the-imm-101-015-trial/

Dosage: “All patients received one 1.0 mg (0.1 mL) dose of IMM 101 intradermally every 2 weeks for the first 3 doses followed by a rest period of 4 weeks, then every 2 weeks for the next 3 doses, and thereafter every 4 weeks. “

“The rapid and complete clinical responses seen in these patients may suggest that IMM-101 is activating a complementary pathway which is synergistic with CPI treatment.”  https://pubmed.ncbi.nlm.nih.gov/30107850/ 

IMM-101 is a safe Treatment – It has been used for 20 years in clinical trials:

IMM-101Published clinical safety trial https://ascopubs.org/doi/10.1200/JCO.2022.40.16_suppl.9554 

Heat killed Mycobacterium vaccae – (the early version of IMM-101) lots of beneficial immune effects https://pmc.ncbi.nlm.nih.gov/articles/PMC10049321/

IMM-101 is the upgraded version that now uses mycobacterium obuense instead of mycobacterium vaccae for manufacturing purposes. 

IMM-101 Needed to activate your Gamm-Delta – the main ones that attack cancer

IMM-101 and Vitamin D stabilises the spread of cancer:

Gamma-Delta Cells the most important in fighting cancer, these are boosted by IMM-101

Innate Immune System needed for cancer. IMM-101 Boosts it:

Cancer switches your immune System off, IMM-01 switches it back on:

Only half the radiation dose needed when using IMM-101

All the bureaucrat Lawrence Tallon has to do is to sign is a form to release IMM-101 under Phase 4 trial status, so it’s the will of the bureaucrat that’s the issue. I need you, I need everyone to pressurise those with the pens to sign this off. Not just in the UK:  Ireland, Mexico , USA, Italy, Spain… the whole world.

IMM-101 origins in the BCG vaccine:

Newspaper article on IMM-101: https://www.theguardian.com/science/2016/sep/06/new-drug-wakes-up-immune-system-to-fight-one-of-deadliest-cancers 

Phase 2 Published studies – clinical trial level:

Conclusions: IMM-101 in combination with nivolumab is safe and shows encouraging antitumor activity in treatment-naïve patients with advanced melanoma. “The Overall Response Rate was 73% in cohort A whereas all pts in cohort B reported progressive disease.”  https://ascopubs.org/doi/10.1200/JCO.2022.40.16_suppl.9554

“Randomised, open-label, phase II study of gemcitabine with and without IMM-101 for advanced pancreatic cancer. Overall Survival was significantly improved from 4.4 to 7.0 months in favour of IMM-101+GEM ” https://pubmed.ncbi.nlm.nih.gov/27599039/ 

https://jitc.bmj.com/content/1/Suppl_1/P215 “Treatment with IMM-101 induces protective CD8+ T cell responses in clinically relevant models of pancreatic cancer”

“One year Overall Survival rate was 90%” https://www.annalsofoncology.org/article/S0923-7534(24)03100-4/pdf 

“Studies with IMM-101 showed the vaccine’s ability to stimulate cancer patients’ immune systems to help kill cancer cells” https://go.drugbank.com/drugs/DB15850

List of published papers on IMM-101: https://pubmed.ncbi.nlm.nih.gov/?term=IMM-101&sort=date&ac=yes

IMM-101 trial is limbo: https://www.cancerresearchuk.org/about-cancer/find-a-clinical-trial/a-study-of-imm-101-and-nivolumab-for-people-with-advanced-melanoma

IMM-101 trial is limbo: IMM-101 trial is limbo:https://www.cancerresearchuk.org/about-cancer/find-a-clinical-trial/a-trial-looking-new-type-immunotherapy-cancer-pancreas-cannot-be-removed-with-surgery#undefined

IMM-101 wakes up and activates the innate immune system so it can fight the cancer: “A suspension of heat-killed Mycobacterium (M.) obuense, a non-pathogenic and naturally-occurring bacterium, with potential immunomodulating and antineoplastic activities. Upon intradermal administration of heat-killed M. obuense IMM-101, the bacteria are able to activate the innate and adaptive immune system. Specifically, IMM-101 activates immature dendritic cells (DCs), which leads to the induction of a type-1 immune response (cDC1). This may induce a cytotoxic T-lymphocyte (CTL)-mediated immune response against cancer cells. It also activates macrophages and natural killer cells (NKs), gamma delta T (gdT) cells, and induces a type 1 T helper (Th1) cell immune response against cancer cells.” https://www.cancer.gov/publications/dictionaries/cancer-drug/def/heat-treated-bacterium-mycobacterium-obuense-imm-101 

https://jitc.bmj.com/content/2/Suppl_3/P54  “A heat-killed preparation of mycobacterium obuense can reduce metastatic burden in vivo”

https://jitc.bmj.com/content/2/Suppl_3/P83 “IMM-101, an immunotherapeutic agent in clinical development as an adjunctive treatment for pancreatic cancer”

Innate immune system is switched-off during cancer (IMM-1010 switches it back on : “Natural killer (NK) cells play an integral role in the immune surveillance of cancers and in defense mechanisms against microbial infections³. NK cells can potentially eliminate tumor cells via receptor-ligand interactions, by releasing cytotoxic granules containing perforin and granzyme, through death receptor-mediated pathways, and secreting a range of effector molecules, such as interferon (IFN)-γ and tumor necrosis factor (TNF)-α³. However, in several types of human tumors, the number of NK cells is lower than that in healthy control tissue, and the cells themselves become dysfunctional, thus rendering NK cell-mediated tumor surveillance ineffective³. https://www.cancerbiomed.org/content/early/2023/08/25/j.issn.2095-3941.2023.0215  

This is Google’s AI on “heat killed mycobacterium obuense”:

• IMM-101 is a suspension of heat-killed Mycobacterium obuense. 

  Immunomodulatory Activity:

  IMM-101 is known for its immunomodulatory properties, meaning it can activate and stimulate the immune system. 

  Mechanism of Action:

  • Activation of Dendritic Cells: IMM-101 activates immature dendritic cells (DCs), which are important antigen-presenting cells that bridge the innate and adaptive immune systems. Type-1 Immune Response: It induces a type-1 immune response, which is characterized by the production of cytokines that help fight off infections and cancer. Cytotoxic T-Lymphocyte (CTL) Response: IMM-101 can induce a cytotoxic T-lymphocyte (CTL)-mediated immune response, where T cells specifically target and kill cancer cells. Activation of Macrophages and Natural Killer Cells (NKs): It also activates macrophages and natural killer cells (NKs), which are other important components of the immune system that can destroy cancer cells. 
    Clinical Trials:

    IMM-101 has been evaluated in clinical trials for various cancers, including melanoma, pancreatic cancer, and colorectal cancer – (The large multi-centred Phase 3 trial has been closed down as it was planned and budgeted for 6 months and “ethics committees” bled it out with admin requests and delays till 18 months and the company ran out of money)
    Potential Benefits:
    Reduced Metastatic Burden: Studies suggest that IMM-101 can reduce the metastatic burden of tumors, meaning it can help prevent cancer from spreading. Immune System Activation: IMM-101 can activate and stimulate the immune system, potentially leading to a more effective response against cancer. 
    Safety and Efficacy:

    A study combining stereotactic body radiotherapy with IMM-101 vaccination for locally advanced pancreatic cancer demonstrated safety and immune-stimulatory effects.

    Even Google knows it is safe and effective !

This IMM-101 phase 3 trial should have been completed in 2022 but the ethics committees in the various locations requested endless paperwork that meant the company ran out of money ad the trial stopped https://biocanrx.com/ct9-auer 

müd+

https://mud-plus.com/

This is a soft gel supplement capsule that contains the earlier version of IMM-101: The heat killed Mycobacterium vaccae; https://pmc.ncbi.nlm.nih.gov/articles/PMC10049321/

Instead of an Intradermal injection, this is instead swallowed and these dead bacteria Mycobacterium vaccae stimulate the Peyer’s patches in the small intestine, particularly the ileum, which are lymph “organs” to stimulate a similar Innate immune wake up call response – but at a lower amount – hence the IMM-101 Injection is best for people in emergency situations – cancer patients. Despite instructions on the packet that you take one a day, the word on the street is that you take 7 in one day, in one go on a weekly basis to get the kick start innate immune system reaction you are after.

There are other “soil-based” probiotic supplements that might have a similar effect – have a google. “A study on 223 patients carried out in 1993 showed that combination therapy including radiation and treatment with heat-killed L. casei strains (LC9018) and improved the induction of immune response mechanisms against cancer cells thereby enhancing tumor regression in patients with carcinoma of the uterine cervix [69] ” https://pmc.ncbi.nlm.nih.gov/articles/PMC6586914/ . It also protects the body against radiation damage: “Radiation-induced leukopenia was significantly less severe (P < 0.05) in the LC9018-combined group than in the radiation-alone group, suggesting that this agent might help to prevent leukopenia during radiation therapy.” https://pubmed.ncbi.nlm.nih.gov/8364872/ 

Low dose Naltrexone (LDN)

Low Dose Naltrexone (LDN): When a patient gets cancer (TB, AIDS) the same – maybe others, the Innate Immune system is switched off either by the cancer itself or by some previous agent. For me, I got the first wave of Covid and it knocked the stuffing out of me. When I recovered, it felt it had aged some part of me by a decade or two. Maybe this feeling I had was my innate immune system now being that of a 70 year old (I was 52 back in 2020). But the other part of the immune system – the adaptive immune system – the clever sophisticated side is still working well. So when you have cancer, the body uses what it has left – this adaptive immune response – The problem is the adaptive immune system is like a bunch of lace-wearing sword fencers with their foil swords being used in a medieval battle field – they are useless – actually they are worse than useless. When the adaptive immune system gets to work it causes inflammation – which is exactly what the cancer wants in order to spread. Low dose Naltrexone (LDN) dampens down this systemic inflammation as well as switching off the IL6 gene activity of the cancer – the “cancer growth factor” and so stops the cancer from spreading. This drug has been used for years at it’s full dose 50MG and is very safe. Taken at 1/10th the dose 4.5MG – but no more as it does not work at higher doses – this is super safe !

I have asked for this on prescription, my GP has said no and tells me to ask my oncologist who can’t be reached. I am not asking here for some super shot that costs £100,000 per injection, I’m asking for a simple 1/10th of a pill that is safe and can stop my cancer spreading as shown in published papers. I hear that other patients can’t get it either. We need to join together to change this appalling situation. 

If you get a private prescription for Low dose Naltrexone the instructions will likely say start at 0.5ML 3 times a week and build up 0.5 ML each week till you get to 4.5 ML – this is a very cautious schedule and may be suited for a delicate CFS/ME patient, but with cancer you will want to build up a lot faster: maybe start at 1ML day 1 then 2 ML day 2, then 2.5ML day 3 then 3.5ML day 4.0 ML day 5 and 4.5ML day 6. If you react to it take a couple of days off and drop down the dose a bit and re-build up a bit slower. 

Low Dose Naltrexone (LDN) works best when it is combined serially with CBD oil – https://www.spandidos-publications.com/10.3892/or.2022.8287 so protocols like “LDN first thing in the morning, then 2 doses of CBD later in the afternoon and evening… but while LDN at 4.5MG remains a daily thing, it is super important that the CBD is a 3 days on, 3 days off pattern – it is on the 3 days off that the cancer cells die, if you don’t have the time off, they don’t die.- it’s like they’ve got hooked on the CBD and going cold-turkey kills them” . “In a study conducted by Massi et al., it was shown that cannabidiol (CBD), like LDN, can modulate the enzymatic pathways of proteins associated with apoptosis processes [103]. Based on this, Liu et al. conducted studies on human lung cancer cell lines A549, human colorectal cancer HCT116, and in vivo models with xenografts derived from these cell lines in mice, aiming to determine whether polytherapy using LDN and CBD could be applicable in the pharmacotherapy of these cancers. It was shown that combined treatment with LDN and CBD did not have a significant impact on the number of proliferating cells; however, sequential exposure of cells first to LDN and then to CBD had a significant effect on reducing the number of live cells and colony count. Additionally, sequential therapy with LDN and CBD caused a significant increase in cell sensitivity to (Chemo) oxaliplatin applied in suboptimal doses. Similarly, the mass and volume of tumors in mice were reduced after applying the described therapy scheme [104]. https://pmc.ncbi.nlm.nih.gov/articles/PMC10968813/

“Combination of cannabidiol with low‑dose naltrexone increases the anticancer action of chemotherapy in vitro and in vivo” and the order of which gets taken first matters: “There was a 35% reduction in cell numbers when using LDN before using CBD compared to a 22% reduction when using CBD before LDN. The two agents also sensitised cells to chemotherapy as significant decreases in cell viability were observed when they were used before chemotherapy” https://pubmed.ncbi.nlm.nih.gov/35179218/ 

Low Dose Naltrexone (LDN) also works well with Alpha Lipoic Acid: “After only a few treatments of IV α-lipoic acid and IV vitamin C, his symptoms began to improve, and the patient regained his baseline weight. His energy and outlook improved, and he returned to work. The patient had stable disease with disappearance of the signs and symptoms of stage IV RCC, a full 9 years following diagnosis, with a gentle integrative program, which is essentially free of side effects. As of November 2017 the patient feels well and is working at his full-time job.”:  https://pubmed.ncbi.nlm.nih.gov/29258346/  

Hydroxycitrate , Alpha Lipoic Acid & LDN amazing results: https://www.youtube.com/watch?v=rwhwv4FjsQc

Tumor regression with a combination of drugs interfering with the tumor metabolism: efficacy of hydroxycitrate, lipoic acid and capsaicin. https://pubmed.ncbi.nlm.nih.gov/22797854/ 

https://pubmed.ncbi.nlm.nih.gov/33337537/  “Low-dose naltrexone targets the opioid growth factor-opioid growth factor receptor pathway to inhibit cell proliferation”

berkson-et-al-2006-the-long-term-survival-of-a-patient-with-pancreatic-cancer-with-metastases-to-the-liver-after  “The key therapeutic agents were intravenous-lipoic acid (ALA) 300 to 600 mg 2 days per week and low dose naltrexone (LDN), 4.5 mg at bedtime. In addition, a triple antioxidant regimen consisting of oral ALA(600 mg/d), selenium (200 g 2 times per day), and silymarin  (Milk Thistle) (300mg 4 times a day ) was added to scavenge the products of oxidative stress that inevitably result from any serious chronic medical disorder. The overall prognosis for patients with carcinoma of the pancreas is poor: the average length of survival after diagnosis ranges from 3 to 6 months.1 Surgical resection is generally not an option for people with metastatic pancreatic  cancer, and patients with advanced metastatic disease rarely survive more than a few months. That J.A. has had comparatively stable disease for more than a 3-year period is a remarkable clinical finding and prompts this report. “

Low Dose Naltrexone (LDN) also works well with vitamin D https://pubmed.ncbi.nlm.nih.gov/25284545/ “It has been determined that the level of the endogenous opiate methionine-enkephalin is increased by LDN. Met-enkephalin is involved in regulating cell proliferation and can inhibit cancer cell growth in multiple cell lines. Increased met-enkepahlin levels created by LDN thus have the potential to inhibit cancer growth in humans.

LDN is the “tonic water” of the anti-cancer world – it mixes with all sorts “Low-Dose Naltrexone as an Adjuvant in Combined Anticancer Therapy. The opioid growth factor (OGF) and its receptor, opioid growth factor receptor OGFr, serve as a tonically active inhibitory axis regulating cell proliferation in normal cells and a variety of cancers, including human ovarian cancer. Blockade of OGF and OGFr with the nonselective opioid receptor antagonist naltrexone (NTX) upregulates expression of OGF and OGFr. Administration of a low dosage of NTX (LDN) blocks endogenous opioids from opioid receptors for a short period of time (4-6 h) each day, providing a window of 18-20 h for the upregulated opioids and receptors to interact. Results: OGF and LDN markedly reduced ovarian tumor burden (tumor nodule number and weight). The mechanism of action was targeted to an inhibition of tumor cell proliferation and angiogenesis; no changes in cell survival were noted. https://pmc.ncbi.nlm.nih.gov/articles/PMC10968813/#B107-cancers-16-01240 

“Low-dose naltrexone (LDN) exhibits antagonistic action against the opioid growth factor receptor (OGFr), whose signaling is associated with the survival, proliferation, and invasion of cancer cells. The mechanism of action of LDN depends on the dose and duration of the OGFr blockade, leading to a compensatory increase in the synthesis of the opioid growth factor (OGF), which has an inhibitory effect on carcinogenesis. Numerous studies on in vitro and in vivo models provide evidence of LDN’s positive impact on inhibiting the OGF–OGFr axis in cancers. LDN’s unique mechanism of action on cancer cells, lack of direct cytotoxic effect, and immunomodulating action form the basis for its use as an adjuvant in chemotherapy and immunotherapy of cancerous lesions.” Low-dose naltrexone (LDN), ranging from 1 to 5 mg, causes a transient blockade of opioid receptors, which results in the “up-regulation” of the endogenous opioid system [44] (Figure 2). LDN exhibits strong, transient blocking action on the Opioid Growth Factor Receptor OGFr [45]. In experimental models, LDN competes with the opioid Growth Factor OGF for binding sites, and after a short period of blockade, through feedback, promotes the release of endogenous opioid peptides as a result of compensatory up-regulation of the OGF–OGFr axis. Subsequently, the use of LDN leads to an increase in the expression of µ, δ, and OGFr opioid receptors [46,47,48], resulting in DNA replication inhibition, and thus limiting the proliferation of cancer cells [49]. LDN can increase the phagocytic ability of macrophages, induce enhanced interactions between CD4+ T lymphocytes and macrophages, and stimulate the cytotoxic activity of NK cells [54]. LDN, applied in animal experimental models, increased the percentage of CD8+ T lymphocytes, which, along with NK cells, dominate the immune response directed against cancer cells through the synthesis of pro-inflammatory cytokines [55]. LDN enhances the production of immunoglobulin G2a (IgG2a) and interferon-γ (IFN-γ), which are responsible for the enhanced proliferation of Th1 lymphocytes. Exposure of lymphocytes to LDN results in an increased synthesis of pro-inflammatory cytokines, such as interleukins-2 (IL-2), interleukins-4 (IL-4), interleukins-6 (IL-6), and IFN-γ, by these cells [56]. Zijain and colleagues demonstrated that LDN induces the transition of macrophages from the M2 to the M1 type, which triggers the mobilization of these cells to secrete higher concentrations of tumor necrosis factor α (TNF-α), interleukin-12 (IL-12), and IL-6, as well as lower concentrations of interleukin-10 (IL-10) [57]. This mechanism is extremely important from an oncology perspective, as M2 macrophages significantly secrete interleukin-10 (IL-10), which promotes carcinogenesis in abnormal cells. The induction of M2 to M1 macrophage transition by LDN leads to reduced levels of IL-10. Moreover, M1 macrophages secrete cytokines that promote the release of reactive oxygen species and NO, having a direct cytotoxic effect on cancer cells. There is evidence in scientific studies that an increased number of M1 macrophages is associated with better prognoses for cancer patients [58,59,60]. The transient blockade of OGFr by LDN enhances the activity of endogenous opioid peptides, promoting the proliferation of B lymphocytes [61]. LDN mobilizes immune cells to act against pathogens in the early stages of the disease, suggesting its potential effectiveness in cancers caused by oncogenic viruses, such as cervical cancer associated with human papillomavirus (HPV) infection [62]. https://pmc.ncbi.nlm.nih.gov/articles/PMC10968813/#B108-cancers-16-01240

“Low dose naltrexone (LDN) enhances maturation of bone marrow dendritic cells” https://pubmed.ncbi.nlm.nih.gov/24455776/ 

“LDN enhances maturation of BMDCs as evidenced by 1) up-regulating the expression of MHC II, CD40, CD83, CD80 and CD86 molecules on BMDCs; 2) down-regulating the rates of pinocytosis and phagocytosis accompanied by the results of decreased ACP, and FITC-dextran bio-assay; 3) mounting potential of BMDCs to drive T cell; and 4) inducing secretion of higher levels of IL-12 and TNF-α.”  https://pubmed.ncbi.nlm.nih.gov/30286124/

“Low Doses Naltrexone (LDN): The Potential Benefit Effects for its Use in Patients with Cancer. (LDN) acts as an Opioid Growth Factor receptor (OGFr) antagonist and the Opioid Growth Factor – Opioid Growth Factor receptor (OGF-OGFr) axis is an inhibitory biological pathway present in human cancer cells and tissues, Clinical trials have proposed a unique mechanism(s) allowing LDN to affect tumors. LDN shows promising results for people with primary cancer of the bladder, breast, liver, lung, lymph nodes, colon and rectum….   https://pubmed.ncbi.nlm.nih.gov/33504322/ 

“naltrexone inhibited production of IL-6… naltrexone can inhibit the non-canonical opioid growth factor receptor, resulting in a decrease in cell proliferation (12–14)”   https://pmc.ncbi.nlm.nih.gov/articles/PMC5504148/ 

Narrow range of dose: Although naltrexone was first employed as a means to support patients with addictive disorders, it was discovered, albeit serendipitously, if used at lower dosages, it could also help with other indications. However, this dose range was very narrow, typically one between 3–5 mg per day for patients. The dosage appeared not to be dependent upon body weight, but more with daily dose, as patients using doses outside of this range commonly reported a loss of activity, which was restored once the dose was re-adjusted to between 3–5 mg/day. More importantly, most of these conditions have a strong inflammatory component and where the effect can be observed directly such as patients with psoriasis, the benefit observed at the commonly used 4.5 mg dose disappears if it is raised to even 6 mg. Reassuringly, however, clinical benefit and activity are quickly restored when the dose is dropped back to 4.5 mg”

“Naltrexone at low doses (LDN) and its relevance to cancer therapy” https://openaccess.sgul.ac.uk/id/eprint/114135/

PDF on LDN: Naltrexone at low doses (2)

https://ldnresearchtrust.org/  – Video in this

Amazon book on LDN:  https://www.amazon.co.uk/Ldn-Book-Little-Known-Naltrexone-Revolutionize/dp/1603586644 

The LDN institute has 3 books . They have a cancer protocol in their LDN Book 3. It is at the end of the book and says to take LDN four days and three days off : https://www.amazon.co.uk/LDN-Book-Low-Dose-Naltrexone/dp/1739107004/ref=sr_1_fkmr1_2 

Only half the dose of standard chemotherapy needed when using LDN and all the chemo toxicity is in the 2nd half

Dosing – how much, and Scheduling – how many days a week, is an issue and looks like it has personal variation – the sort of thing you’d have hoped that 125 years of the multi-Billion cancer research industry might have looked at by now… but no !

Patients who have Lyme disease and large yeast infections need to start slow as the healing can cause a big kick back. Codeine is a problem with LDN as well:

This patient find that varying his dose works for him very well:

Results: “Several case reports demonstrate notable survival durations and metastatic resolutions in patients with late stage cancer when administered an average LDN dose of 3-5 mg/day”.  https://pubmed.ncbi.nlm.nih.gov/33337537/  

LDN can actually be used at slightly higher doses: 2 or 3 4.5mg tablets a day for stroke victims and complex regional pain syndrome:

Lawrence Tallon, head of the UK’s MHRA Medicines and Healthcare products Regulatory Agency, can add cancer to the list of things that LND can be prescribed for today.

Published study on Low dose Naltrexone.(LDN): “Naltrexone at low doses (LDN) and its relevance to
cancer therapy”: Low dose Naltrexone can also boost natural Killer cells – and so possibly Gamm-Delta and other Innate immune cells – though they have not looked at this yet: and it enhances chemo therapy allowing only half the usual does to be used:  “which in turn can sensitize cancer cells to the cytotoxic effects of common chemotherapy agents [21] … the schedule of naltrexone administration was also crucial, with intermittent administration of low-dose naltrexone achieving the greatest anti-tumor response.”  https://openaccess.sgul.ac.uk/id/eprint/114135/1/Naltrexone%20at%20low%20doses%20LDN%20and%20its%20relevance%20to%20cancer%20therapy.pdf

LDN switches off IL6 – the cancer growth factor

LDN, applied in animal experimental models, increased the percentage of CD8+ T lymphocytes, which, along with NK cells, dominate the immune response directed against cancer cells through the synthesis of pro-inflammatory cytokines [55]. LDN enhances the production of immunoglobulin G2a (IgG2a) and interferon-γ (IFN-γ), which are responsible for the enhanced proliferation of Th1 lymphocytes. Exposure of lymphocytes to LDN results in an increased synthesis of pro-inflammatory cytokines, such as interleukins-2 (IL-2), interleukins-4 (IL-4), interleukins-6 (IL-6), and IFN-γ, by these cells [56]. Zijain and colleagues demonstrated that LDN induces the transition of macrophages from the M2 to the M1 type, which triggers the mobilization of these cells to secrete higher concentrations of tumor necrosis factor α (TNF-α), interleukin-12 (IL-12), and IL-6, as well as lower concentrations of interleukin-10 (IL-10) [57]. This mechanism is extremely important from an oncology perspective, as M2 macrophages significantly secrete interleukin-10 (IL-10), which promotes carcinogenesis in abnormal cells. The induction of M2 to M1 macrophage transition by LDN leads to reduced levels of IL-10. Moreover, M1 macrophages secrete cytokines that promote the release of reactive oxygen species and NO, having a direct cytotoxic effect on cancer cells. There is evidence in scientific studies that an increased number of M1 macrophages is associated with better prognoses for cancer patients [58,59,60] https://pmc.ncbi.nlm.nih.gov/articles/PMC10968813/#B107-cancers-16-01240

LDN turbo boosts the innate immune system: “We have found that LDN enhances function of macrophage as confirmed by up-regulating MHC II molecule and CD64 on macrophage while down-regulating CD206 expression. Furthermore the productions of TNF-α, IL-6, IL-1β, increased significantly. Macrophages in LDN treated group performed the enhanced phagocytosis. Therefore it is concluded that LDN could promote function of macrophage and this work has provided concrete data of impact on immune system by LDN. Especially the data would support interaction between CD4+T cell and macrophage in AIDS treatment with LDN in Africa (LDN has already been approved in Nigeria for the use in AIDS treatment). https://pubmed.ncbi.nlm.nih.gov/27561742/

Dr. Ian S. Zagon’s work on LDN https://ldnresearchtrust.org/sites/default/files/dr-zagon.pdf 

 

Webinar on LDN – very comprehensive: https://www.youtube.com/watch?v=5pxdq64kAmw

Vitamin D:

Calcifediol – this is fast acting vitamin D. No cancer treatment works unless the patient has vitamin D levels of 100 ng/ml or upwards. If a patient is low and supplements with D3 then a) the D3 takes around 3 weeks+ to be converted by the liver to a useable form – those 3 weeks can be the difference between life and death. And the patient, being very ill, might no be able to achieve this conversion so well anyway – lots of patients take High level of vitamin D and their levels do not rise. Calcifediol is what the liver is trying to convert Vitamin D3 to it to. A simple Vitamin D blood test and supplementation immediately by Calcifediol, and backed by D3 supplementation, when it is too low, is what is needed for any treatment to be effective as Calcifediol takes only a couple of hours to get into the system, so the vitamin D form you are ready to use now to fight your cancer is immediately available. I have asked my GP for this test and prescription should it be needed. So far no reply – time is of importance ! I am not the only one. Isolated in the GP’s surgery I am on my own against the might of the medical world. In this state I am a squashable bug in terms of power relations. Joined together as The Patient Power Association we are a global voice – a power block with a full seat at the table.

Adequate Vitamin vitamin d 100 nmol/L (=30 ng/mL) + needed for any cancer treatment to work: “I cannot believe it, the only people who respond to Chemo are the only people who have normal Vitamin D, it is Black and White”

Adequate Vitamin vitamin d 100 nmol/L (=30 ng/mL) needed for any cancer treatment to work. “It is pointless treating cancer patients until you’ve got their Vitamin D levels up to adequate.

Vitamin D Protocol used at St George’s hospital, London

Unless you have adequate Vitamin D Levels, your NK Natural Killer white blood cells can’t attack and destroy the cancer:

Update: I told my consultant about the approach St George’s now do and showed the video clip on my phone. He agreed to give me a vitamin D test there and then. It came back today my results were a nice large 187 nmol/l = 74.8 ng/dL. I don’t supplement with D3, but for the past few months I’ve been chopping up mushrooms and putting them, gill upwards, under a UV-B lamp designed for a lizard from a pet shop for an hour. Mushrooms done like this produced huge amounts of natural vitamin D and also it looks like it absorbs vert well as well – which does not always happen with the tablets.

The IMM-101 and the Low Dose Naltrexone (LDN) work well together as the Gamma-Delta cells that are produced as a result of the IMM-101 need to get at the cancer. The cancer will be protected by inflammation and the Low Dose Naltrexone (LDN) dampens down the inflammation allowing the Gamm-Delta cells to get at and kill the cancer cells.

 

Other Super useful things:

Pacreatic Enzymes and B17 Apricot Kernels:

The important thing about taking pancreatic enzymes is that you take them on an empty stomach or in an enteric capsule – one that does not dissolve in the stomach – otherwise they will be used just to digest the food you eat. I take mine at 6am in the morning and go back to bed. In layman’s terms, the idea of them is that they attack the outside protective layer of the cancer cells and so weaken them for attack by your immune system and every other therapy you do. 

Pancreatic enzymes: Trypsin and Amylopsin (pancreatic amylase): ” In 1902, in an article in The Lancet, Beard ¹ proposed that the answer to questions about the origin of cancer could be found in his own field, embryology. He observed that the early stage of the placenta, the trophoblast, looked and acted much like a cancer as it invaded the uterus and created a blood supply. He reported the presence of “vagrant germ cells” in the tissues of embryos far from the placenta, and suggested that these could become like invasive trophoblast cells, forming the nidus of development of cancer in the future.^2,3 In normal development, trophoblast cells differ from cancer in 1 key respect. At a certain point in development—Beard claimed at 56 days in humans—the trophoblast stops invading, changing in character from a poorly differentiated, angiogenic tissue into the mature placenta. Beard looked for the signal that induced this transformation, believing that if he found it, he might also find a treatment for cancer. He reported that in a number of mammalian species, this change takes place when the fetal pancreas begins manufacturing proteolytic enzymes. ⁴ Beard ⁵ then tested his theory in a mouse model of cancer, Jensen’s mouse tumor. After injections of the commercially available pancreatic enzyme trypsin, the tumor in a treated mouse was much smaller than that in an untreated control.”

“Kelley’s protocol included dietary modification and coffee enemas, as well as large amounts of oral pancreatin. After he regained his health, others with cancer sought him out, and his practice gradually migrated from orthodontics to controversial cancer treatment.”

A collection of case reports published in 2007 included a patient who had applied and been refused admission to the clinical trial mentioned above. In December 2000, computed tomography showed a 3.4 cm mass in the head of the pancreas; biopsy February 2001 demonstrated poorly differentiated adenocarcinoma. The pathology slides were then sent to the Mayo Clinic, where the diagnosis was confirmed. She was denied entry to the clinical trial because her disease was considered resectable (i.e. a tumor can be completely removed with surgery) , though she had refused surgery multiple times. She then embarked on the enzyme-based nutritional protocol outside of the clinical trial. She has now survived more than 20 years since biopsy. She has never received surgery, chemotherapy, or radiation. Patient 2: He subsequently began an enzyme-based nutritional program in April 2016. A scan 2 months later showed regrowth of the liver lesion and a new pulmonary mass. The liver lesion was resected in July 2016 and reported to be “Metastatic nodule of colorectal-type adenocarcinoma with no residual viable tumor identified.” Since then, the patient has had no treatment besides his nutritional program. On subsequent scans, the pulmonary lesion gradually disappeared, to the astonishment of his oncologist. At last contact December 2021, he was alive and well, working at a demanding job while also continuing his treatment protocol. Recent scans of the thorax and abdomen showed no sign of disease.”

“The 2 most recently published case reports illustrate the importance of adherence. ²⁸ The first patient was diagnosed with colon cancer that was resected May 2014. After his carcinoembryonic antigen began to rise, he began chemotherapy in early 2015. A new liver lesion developed in June 2015, and he stopped chemotherapy. After further growth, the liver lesion was removed in February 2016 and confirmed to be metastatic colon cancer, extending to the margin of resection. He subsequently began an enzyme-based nutritional program in April 2016. A scan 2 months later showed regrowth of the liver lesion and a new pulmonary mass. The liver lesion was resected in July 2016 and reported to be “Metastatic nodule of colorectal-type adenocarcinoma with no residual viable tumor identified.” Since then, the patient has had no treatment besides his nutritional program. On subsequent scans, the pulmonary lesion gradually disappeared, to the astonishment of his oncologist. At last contact December 2021, he was alive and well, working at a demanding job while also continuing his treatment protocol. Recent scans of the thorax and abdomen showed no sign of disease.

The second patient developed neurological symptoms leading to the discovery of masses in his lung and brain in February 2014. The brain mass was resected and found to be metastatic adenocarcinoma, most likely non-small cell lung cancer. He received radiation to the brain, then embarked on a self-designed nutritional program. With this, his lung masses enlarged and he developed recurrent brain lesions.

In January 2015, he began the enzyme-based nutritional program, but he also underwent more radiation to the brain due to concerns about incipient herniation. In August 2015, scans showed resolution of the pulmonary masses previously seen, and stable findings in the brain. He never had radiation targeting his lung, or systemic therapy.

He did well until June 2018, when he developed focal seizures and weakness. Scans of the chest showed no evidence of disease; magnetic resonance imaging of the brain showed recent hemorrhage in the mass that had been present since radiation. His physicians concluded that he had residual radiation damage and acute clinical deterioration due to bleeding. Unfortunately, he was left with hemiparesis that made it hard for him to take care of himself; he lived alone, with no outside support. He discontinued his enzyme-based nutritional program, and 2 years later his disease recurred. He died in May 2020, more than 6 years from diagnosis. In comparison, in a case series of patients with a similar initial presentation, median survival was around 9 months. ²⁹     https://pmc.ncbi.nlm.nih.gov/articles/PMC9083047/

“In a 2001 article, Sakalova et al ³⁰ reported prolongation of life in patients with multiple myeloma treated with Wobe-Mugos E, an oral enteric-coated combination of papain, trypsin, and chymotrypsin(that is (“out of stock everywhere I’ve looked” !) . In this retrolective cohort study, Stage III patients who received Wobe-Mugos E in addition to chemotherapy survived 83 months, compared with 43 months in the control group who received only chemotherapy. Reviews of other studies using Wobe-Mugos have been published elsewhere. ¹⁴ In a 2008 article, Wald ³¹ reported positive results in mouse experimental tumor models with rectally administered enzymes in composition and proportions similar to Wobe-Mugos.”

“A formulation of pancreatic pro-enzymes provides potent anti-tumour efficacy: We present a combination of the two pro-enzymes Trypsinogen and Chymotrypsinogen A with potent in vitro and in vivo anti-tumour efficacy. A synergetic anti-tumour effect for Trypsinogen and Chymotrypsinogen A was determined at a ratio 1:6 (named PRP) “ https://pubmed.ncbi.nlm.nih.gov/29070896/  

Trypsin, chymotrypsin, and papain have been documented in a series of animal experimental tumor models. The experimental evidence demonstrates a significant inhibition of growth of both the primary tumor and the metastatic disseminations. https://pubmed.ncbi.nlm.nih.gov/19116225/ 

“This pilot study suggests that an aggressive nutritional therapy with large doses of pancreatic enzymes led to significantly increased survival over what would normally be expected for patients with inoperable pancreatic adenocarcinoma”.https://pubmed.ncbi.nlm.nih.gov/10368805/ 

Tumors in the Pancreatic enzyme extract-treated group were significantly smaller than in the control group. All mice in the control group showed steatorrhea, hyperglucosuria, hyperbilirubinuria, and ketonuria at early stages of tumor growth, whereas only a few in the treated group showed some of these abnormalities at the final stage The treatment with Pancreatic enzyme extract significantly prolongs the survival of mice with human PC xenografts and slows the tumor growth https://pubmed.ncbi.nlm.nih.gov/15097858/   

“Here we investigate in vitro and in vivo the effects of a pancreatic (pro)enzyme mixture composed of Chymotrypsinogen and Trypsinogen (PRP) on CSCs  We can conclude that they reduce the Cancer Stem Cells population, tumour initiation and decreases fibrotic tissue in tumours. ” https://pmc.ncbi.nlm.nih.gov/articles/PMC6684636/ 

Chymotrypsinogen and Trypsinogen acted on Cancer Stem Cells and resulted in a significant decrease of ALDEFLUOR and specific pancreatic CSC markers (CD 326, CD 44 and CxCR4), they also nhibited primary and secondary sphere formation. Also (i) epithelial-mesenchymal transition (EMT) inhibition; (ii) CSCs related genes suppression; (iii) enhanced expression of tumour suppressor genes; (iv) downregulation of migration and metastasis genes and (v) regulation of MAP Kinase Signalling Pathway. Finally, in vivo anti-tumor xenograft studies demonstrated high anti-tumour efficacy of PRP against tumours induced by BxPC3 also  impaired engrafting of pancreatic CSC’s tumours in nude mice and displayed an antigrowth effect toward initiated xenografts. We concluded that (pro)enzymes treatment is a valuable strategy to suppress the CSC population in solid pancreatic tumours. https://pubmed.ncbi.nlm.nih.gov/31388092/ 

“Alpha-Amylase Inhibits Cell Proliferation and Glucose Uptake in Human Neuroblastoma Cell Lines” https://pmc.ncbi.nlm.nih.gov/articles/PMC9343180/  

You can also get freeze dried ground up pancreas in capsules. 

Apricot Kernels: Amygdalin from Apricot Kernels Induces Apoptosis and Causes Cell Cycle Arrest in Cancer Cells: An Updated Review”.  https://pubmed.ncbi.nlm.nih.gov/29308747/ ” Of all the social media groups I’ve joined over the past few weeks since this all started : FenBen, Ivermectin… it is the B17 Apricot Kernel groups that have the best patient stories by a long shot: Lots of comments like “I do enzymes for my pancreas 2-3 a meal and I eat 8-10 seeds twice a day ! My cancer is gone now” “It is recommended to slowly work your way up to 10 seeds 4x/day for a total of 40 seeds for active cancer patients. Take pancreatic enzymes on an empty stomach to break down the protein walls of cancer cells. Do not take vitamin C at the same times as the seeds because it will burn up the Amygdalin and it will be useless” and practical hints about not over doing it as it can lower blood pressure o that is something to keep an eye one and don’t do a silly amount in one go : “Last 2 weeks I was adding 10-15 kernels to smoothies three times a day without issue. Yesterday I was out the entire day. I made a smoothie with 35 ( or more) kernels with parsley and pineapple after dinner. Immediately I was having serious heart palpitation.” It even works for dogs: “3 kernels for every 10lb of weight 3 times a day my dog was 40lb and she was having 36. And also blackseed oil 3 times a day it was gone in 3 weeks. Make sure the blackseed oil has over 4% thymoquinone in it. And it’s 1 teaspoon every 20lb weight 3 times a day.”

Protocols vary a little bit: this is one I am trying to keep to: “Three to six seeds per waking hour (for a total of 60 seeds per day) should be chewed and consumed every day for the first month. The pancreatic enzyme that is found in the pineapple and papaya is necessary to burn away the cancer cell wall for the B17 to more effectively get in and destroy the malignant cell. Pancreatic enzymes can be purchased at any health food store, just ask. After the initial month of treatment a maintenance dose of three 500mg tablets per day should be maintained”. You can buy apricot kernel capsules and you can take 500mg after a meal and this will mean you only need to chump on 30 kernels a day chumping on lots of them is important as the mouth is a great place to absorb the B-17. Though as you can see Dave just did 25 kernels a day. I aim for 60 but as I take a break around meal times and… I end up at about 30 well chewed mulched kernels a day plus one apricot kernel capsule. I am left with a small mouthful of the brown outer later of the seed, spit this out if you swallow lots of them this might irritate your gut a bit. So the enzymes on their own are looking effective, as are the apricot kernels… and they work super well together. I take pineapple and papaya enzymes as well as pork based ones that are freeze dried pancreas. 

“Current there are several lines of in vitro evidence suggesting that amygdalin and its synthetic analogue, laetrile, possess anticancer properties, and previous and upcoming in vivo animal studies appear to confirm their anticancer properties.” https://pmc.ncbi.nlm.nih.gov/articles/PMC10531689/ 

Zinc turbo charges Amygdalin  – B17 : “Amygdalin is a natural anti-cancer agent, which can be used for the treatment of hepatocellular carcinoma. It promotes apoptosis via the intrinsic cell death pathway (the mitochondria-initiated pathway) and cell cycle arrest at G/M. The potency of amygdalin in HepG2 treatment increased significantly by the addition of zinc.” https://pmc.ncbi.nlm.nih.gov/articles/PMC10531689/#B55-ijms-24-14270 

Metformin turbo charges Amygdalin   “combination has a promising effect when compared to amygdalin or metformin alone as it is more cytotoxic and have higher ability for induction of apoptosis and arresting cell cycle” https://pubmed.ncbi.nlm.nih.gov/34536497/ 

Rutin 

“the antitumor effect of rutin is related to interactions with signalling processes such as mixed-lineage protein kinase 3, Wnt, and mitogen-activated protein kinase. Furthermore, it demonstrated that it inhibits cell proliferation and regulates apoptosis”  https://pmc.ncbi.nlm.nih.gov/articles/PMC8416379/ 

CBD oil. 

The “word on the street” – so this is not published work – is that CBD oil is very useful for cancer when done on a 3 day on 3 day off routine for some unknown reason. 5mg to 10mg per day – via drops under the tongue. When used on a daily basis it is near useless.  LDN can allow you to have just half the dose and daily CBD oil backs this up nicely when taken afterwards. There is a published study on the LDN (see above) and on the CBD oil (below). Used like this, CBD oil is as effective as the full THC oil without all the problems that brings on it’s own when pulsed (3 days on 3 days off) “… except for brain tumours as the THC oil can cross the blood brain barrier, but combining both can be even more powerful  – again 3 days on 3 days off” 

“CBD did not have abuse potential and caused no harm (3). Studies have shown that in addition to being able to induce cell death directly, it is also capable of interfering with intracellular signalling (4). Alterations to pathways such as the PI3K/AKT/mTOR and the ERK, suggests that CBD can modify the way certain cancer cells react to other treatments. Indeed, studies have shown that combining CBD with conventional chemotherapy such as cytarabine and vincristine can lead to enhanced anticancer activity through modifications to these signalling pathways (5, 6). Furthermore, the sequence in which these drugs are administered can also influence overall activity (5) . Studies have also indicated that in certain leukaemia cell lines, CBD can increase the expression of the cyclin-dependent kinase inhibitor p21^waf1 (6). This increased level appears to be maintained by CBD, which inadvertently impedes cell death. Cytotoxicity can be restored in these cells if the treatment regimen was altered to allow for a temporary cessation of exposure to CBD. Thus, the general efficacy of CBD may also be altered by adapting treatment protocols that include “drug-free” phases”  https://ar.iiarjournals.org/content/38/10/5831 

“Results show a number of cannabinoids could be paired together to generate an effect superior to that achieved if the components were used individually. For example, in HL60 cells, the IC50 values (the lower the values, the more potent the agent) at 48hrs for CBD alone was 8µM and for THC it was 13 µM, respectively; however, if used together, it was 4 µM. Median-effect analysis confirmed the benefit of using cannabinoids in pairs, with calculated combination indices being <1 in a number of cases. The most efficacious cannabinoid-pairs subsequently synergised further when combined with the chemotherapy agents, and were also able to sensitise leukaemia cells to their cytotoxic effects. The sequence of administration of these drugs was important though; using cannabinoids after chemotherapy resulted in greater induction of apoptosis, whilst this was the opposite when the schedule of administration was reversed. Our results suggest that when certain cannabinoids are paired together, the resulting product can be combined synergistically with common anti-leukaemia drugs allowing the dose of the cytotoxic agents to be dramatically reduced yet still remain efficacious. Nevertheless, the sequence of drug administration is crucial to the success of these triple combinations and should be considered when planning such treatments.” https://pubmed.ncbi.nlm.nih.gov/28560402/ 

“The most significant finding of the current study is that removing cells from medium-containing cannabinoids, and allowing them to recover in drug-free medium results in a dramatic increase in cytotoxicity.”  https://ar.iiarjournals.org/content/33/10/4373  

bromelain: 

Bromelain is an extract of Pineapple core, it has super effective anti-inflammatory properties. The proximate cause of cancer is long term inflammation and this is a nice easy cheap safe way to significantly reduce inflammation a lot. Just one capsule a day.

“Bromelain exerts cytotoxic effects in a panel of human gastric and colon carcinoma cells. There are different mechanisms in bromelain-induced cell death. While promoting apoptosis with involvement of the caspase system and extranuclear p53, bromelain also appears to impair cancer cell survival by blocking the Akt pathway and attenuating Bcl2 and MUC1 oncoproteins. ” 800Mg to 1,500 Mg per day on an empty stomach  https://pubmed.ncbi.nlm.nih.gov/23620673/ 

Melatonin: 

“Melatonin reduced the risk of death at 1 yr by 44% . The substantial reduction in risk of death, low adverse events reported and low costs related to this intervention suggest great potential for melatonin in treating cancer.” https://pubmed.ncbi.nlm.nih.gov/16207291/  “We use 60Mg 4 times a day, 180 MG at night:  https://www.youtube.com/watch?v=Roh4lQXneQg 

How does it do this ? Via several methods: Basic mechanisms involved in the anti cancer effect of melatonin https://pubmed.ncbi.nlm.nih.gov/21062257/

“Due to the broad range of melatonin’s actions, the mechanisms underlying its ability to interfere with metastases are numerous. These include modulation of cell-cell and cell-matrix interaction, extracellular matrix remodeling by matrix metalloproteinases, cytoskeleton reorganization, epithelial-mesenchymal transition, and angiogenesis” https://pubmed.ncbi.nlm.nih.gov/27706852/ 

Caynne Pepper:

“Capsaicin inhibits the PI3K/Akt/mTOR axe and modulates autophagy in both LNCaP and PC-3 cells. inhibition of the Akt/mTOR pathway leads to induction of autophagy in many cell types” https://pmc.ncbi.nlm.nih.gov/articles/PMC4811481/#:~:text=Capsaicin%2C%20the%20pungent%20ingredient%20of,cancer%20cells%2C%20including%20prostate%20cancer.

Cousin of Low Dose Naltrexone … Low-Dose NALMEFENE https://www.youtube.com/watch?v=GowsayN0Xkw 

Iodine:

The world and his wife are deficient in this mineral. This is not just of useful for cancer prevention & treatment of the thyroid, it has a role to play in all cancers. Understudied but it obvious that when studied properly it will be found to have a significant say in cancer prevention & treatment. Why wait ? It’s an easy fix to supplement with. A few drops of Lugol’s iodine can be painted on the skin – say top of the leg – and the idea of this is to allow your body to then absorb what it wants, and leave the rest. But that might not be enough and a few drops in water might be needed. Safety note: Lugol’s is administered by drops – small amounts ! A standard dropper contains 1ml of liquid – you use just 1 drop (or 2 or 3), not the whole dropper. Problems have occurred when people read “take two drops” and understand “two full dropper-fulls”   : https://pmc.ncbi.nlm.nih.gov/articles/PMC4495864/ . Stomach and oesophagus irritation can occur at these Ml levels so skin painting is an easy option. Apply every week or so before bed.  

What about covid and the shots ?

I had no shots, but I got hit hard by Covid. It felt very “unnatural”. I came across this a few days ago. it’s a brand new thing called “Augmented NAC” . I gave it a try. I monitor my PH level several times a day and after taking this my urine became very acidic, so I upped my sodium bi-carb to help balance it but it still took two days, while still taking 1 Augmented NAC capsule 3 times a day before this the acidity went down to normal. So I think it cleared out a load of old spike – hopefully that will have an effect on my ability to fight the cancer. I took it for 3 days while I was having a “3 days off” Fenben break so as not to overload my liver. 

USA ones: Ivermectin & Fenbendazole.

These are anti-worming agents. It started of with Joe Tippens: https://x.com/JimmyFalk_55/status/1877737178148606051/video/1 . A vet was testing cancer treatments in mice, the mice all got a worm infestation, so they were treated with  Fenbendazole and it got rid of the parasites… and the cancer ! Cancer cells share similar mechanism to parasites and the Canadians and Americans have been talking about this for nearly 10 years now. Sometimes they fail, so matching UK to USA, thinking – is this because they are low in vitamin D ? Quite possible. Add the UK to the USA approaches: powerful anti-cancer treatments matched with low inflammation, adequate vitamin D levels and the boost the big cancer eating Gamma-Delta and also the Natural Killer cells to be fully activated to attack these weakened cancer cells – it’s a no brainer. It’s a crime against humanity to not use this protocol or at least allow it to be used… today, this morning… this afternoon.. now !

In terms of dosage, in this video he suggests 6 days on, 1 days off, backed by Milk Thistle, or it’s extract silymarin (“Several studies have indicated that silymarin may suppress the proliferation of different tumor cells, such as prostate (18), breast (19), colon (20), ovary (21), lung (22), and bladder (23). “) ,  & Tudca (Take 500-1000 mg of Tudca 1-2 x daily on an empty stomach: “TUDCA suppresses NF-κB signaling and ameliorates colitis-associated tumorigenesis”  https://pubmed.ncbi.nlm.nih.gov/30378164/ )  to help the stress on the liver. But Milk thistle can act a bit like estrogen so hormone-sensitive cancers such as breast cancer, uterine cancer, ovarian cancer might get  worse so stick to the Tudca in these cases, it also causes problems for those with ragweed allergies. Standard Blood tests will show most of these and if they are out of normal range, if they are you need to take a break from the Fenbendazole – maybe for up to 3 months :

ALT (Alanine Aminotransferase) The most specific marker for liver damage – this is the main one to look out for.
AST (Aspartate Aminotransferase) Also signals liver stress, but it’s less specific it can rise with muscle injury too.
ALP (Alkaline Phosphatase) Elevated levels may point to bile duct or gallbladder issues.
GGT (Gamma Glutamyl Transferase) Can be sensitive to medications and detox load.
Total Bilirubin measures how well the liver is clearing waste.

Other items on the standard blood test to look out for:

CRP – C-Reactive protein, the normal range is 0 mg/L to 5mg/L. The higher it is, the higher the level of inflammation in the body. You are not really “out of inflammation” until you are 0.5mg/L or less. This is important as cancer loved inflammation. Vitamin C is very good for a quick reduction in this C-Reactive protein: https://pubmed.ncbi.nlm.nih.gov/18952164/ 

The other ratio to look at in these blood results is the neutrophil-to-lymphocyte ratio, the higher it is the more inflammation. 1.5 is a good ratio, 2.0 is OK. 

I take a Fenbendazole table, crush it in a mortar and pestle, add at least two spoons of plain unsweetened live yoghurt. In less than 2 mins the tablet will have dissolved, I then eat the yoghourt. This is best eaten as a starter before a more substantial main meal that has lots of fat in it. It looks like extra vitamins are needed though to get Fenbendazole to work: “Neither diet supplemented with vitamins alone nor fenbendazole alone caused altered tumor growth as compared with that of controls. However, the group supplemented with both vitamins and fenbendazole exhibited significant inhibition of tumor growth.” https://pmc.ncbi.nlm.nih.gov/articles/PMC2687140/  . The vitamins supplemented were: A,Retinol,D3,Cholecalciferol (prescription only in UK and won’t need if you have adequate vitamin D levels), E,K3,B1,B2,niacin,B6,Pantothenic acid,B12, biotin, Folate – but none in any mega dose, just average health food shop levels.

Ivermectin tablets dissolve in yoghurt in the same way. I take 3 to 5 at a time (12mg each) to spread the dosage during the day.  “These studies show more than a 50% reduction in tumor volumes after ivermectin treatment, which varied from 10 to 42 days of treatment by either oral, intraperitoneal or intratumoral routes (more commonly intraperitoneal). ” https://pmc.ncbi.nlm.nih.gov/articles/PMC5835698/#:~:text=These%20studies%20show%20more%20than,routes%20(more%20commonly%20intraperitoneal). 

Ivermectin is turbo charges with zinc: https://firstmedinc.com/should-take-ivermectin-empty-stomach/

It might though be an idea to follow the 3 day on, 3 day off pattern that works so well with the CBD oil. This is what is used in these case studies: ” Near complete resolution of persistent left renal mass (left image) after initiation of three total doses of nivolumab and FBZ therapy 1 gm three times weekly for several months (right image)., Near complete radiographic response of a 2.0 cm × 1.5 cm aortocaval node (left image) after initiation of alternative therapy including FBZ therapy 1 gm three times weekly , Resolution of 7.5 cm right lateral bladder mass (left image) after treatment with TURBT, Accelerated Methotrexate, Vinblastine, Doxorubicin, and Cisplatin (AMVAC), and concurrent FBZ 1 gm three times weekly (right image).” https://www.scitechnol.com/peer-review/fenbendazole-enhancing-antitumor-effect-a-case-series-2Kms.php?article_id=14307

A large list of studies on Ivermectin, Fenbendazole… and cancer: https://right2try.com/sources.php

The “press pulse” of the metabolic approach below has a similar on-off thing that is important for it’s approach. The 3 days on, 3 days off is also recommended in this published paper here along with other useful ideas like IV Vitamin C and High Pressure Oxygen HBOT – a not on HBOT – lots of cancer patients go to Keto diet. Doing HBOT while in Keto can cause hypoglycaemic shock so it’s best to break Keto for a short while – a day before and during your HBOT session:

Targeting the Mitochondrial-Stem Cell Connection in Cancer Treatment: A Hybrid Orthomolecular Protocol

Short Fenbendazole video:

Fenbendazole published case studies:  https://www.scitechnol.com/peer-review/fenbendazole-enhancing-antitumor-effect-a-case-series-2Kms.php?article_id=14307  

For those with brain cancer, mebendazole can get past the blood brain barrier, so is preferred to Fenbendazole.

Longer Fenbendazole video:

Published paper on  Ivermectin, Mebendazole, Fenbendazole: https://isom.ca/article/targeting-the-mitochondrial-stem-cell-connection-in-cancer-treatment-a-hybrid-orthomolecular-protocol/ 

When I first took Fenbendazole I did 2 X 444MG per day in morning and evening with fatty foot: olive & coconut oil. I tried 6 days on, 1 day off, but I got a lot of constipation so I backed off to 3 days on , 3 days off. I later tried adding half a tea spoon of pectin, twice a day plus “oat fibre” – something the keto people came up with, to some plain fresh yoghurt and this has helped the constipation a lot, allowing me to move up to 3 X 444MG per day… but I’m still undecided about how many days a week, I now crush the tablets and add them to plain yoghurt where they dissolve in a minute . 

The Ivermectin tablets I have dissolve in plain Yoghurt. Ivermectin as an inhibitor of cancer stem‑like cells: “Ivermectin preferentially inhibited the Cancer Stem-like Cell subpopulation in the MDA‑MB‑231 cells and downregulated the expression of genes involved in the maintenance of pluripotency and self‑renewal” https://pubmed.ncbi.nlm.nih.gov/29257278/   

“Ivermectin inhibits tumour metastasis by regulating the Wnt/β-catenin/integrin β1/FAK signalling pathway” 
https://pmc.ncbi.nlm.nih.gov/articles/PMC9641399/

“Ivermectin suppresses tumour growth and metastasis through degradation of PAK1 in oesophageal squamous cell carcinoma”
https://pubmed.ncbi.nlm.nih.gov/32237037/ 

“Ivermectin has powerful antitumor effects, including the inhibition of proliferation, metastasis, and angiogenic activity, in a variety of cancer cells.”  https://pmc.ncbi.nlm.nih.gov/articles/PMC7505114/

Short Ivermectin-cancer video:

Long ivermectin ant-cancer longer  video:

Ivermectin PDF paper to download: “The anti cancer potential of ivermectin”  JLAR_-9 

Ivermectin for oesophageal cancer with not chemo or radiation:

https://rumble.com/v6hphwy-watch-now-dr.-kathleen-rudy-cancer-surgeon-drops-moringa-bombshell.html 

Ivermectin for prostate cancer after being sent to hospice to die:

Another ivermectin & Fenbendazole video:

This is a mini-master class in immunology:

I am a patient, I have stomach cancer, if it is my time to go, then so be it. But if all this is is something that, despite looks and acts horrible, is actually as simple to fix as a worm infestation is, but I’m not allowed to have these treatments due to a Vogel level bureaucrat then this is not acceptable. I do not want to die by bureaucrat. 

Time is of the essence. We need patients on this, the press on this, politicians on this, celebs, sports stars…. the works ! – Current update 28 March 2025 – I have been doing this and I am now currently banned by Twitter (X)… funny eh ?

The two full interviews where the above snippets came from:

The Americans are also joining the “Boost the innate Immune system” approach. Dr Dr Patrick Soon-Shiong of the Chan Soon-Shiong institute for medicine (and also owner of the LA Times)  has developed a “Bio-Shield” booster. He also injects it intradermally, so both Patrick Soon-Shiong and Angus Dalgleish are on a similar page. Patrick’s “Bio-Shield” aims to boost the NK – Natural Killer cells, whereas Professor Dalgleish’s IMM-101 is mainly boosting the more important gamma-Delta – a shot of either in different arms might produce even better results together.

If anyone knows Patrick, Angus is very keen to chat to him, can you let him know: https://tuckercarlson.com/tucker-show-patrick-soon 

Other Cinderella treatments:

Diet:  “A Ketogenic Diet significantly enhances the anti-tumour effect of radiation”:  https://pubmed.ncbi.nlm.nih.gov/22563484/

“Keto Diet during chemotherapy lead to a reduction in tumour size in the Keto Diet compared to the non-Keto diet: tumour size reduction 27mm reduction compared to 6 mm reduction”  https://www.sciencedirect.com/science/article/abs/pii/S0261561420303393

Another fasting video:

You need to block both glucose and Glutamine food sources :

Press Pulse approach by Dr. Thomas Seyfried

https://nutritionandmetabolism.biomedcentral.com/articles/10.1186/s12986-017-0178-2 

The idea of Press-Pulse is you “Press” – provide long term pressure/stress on the cancer by restricting Sugar & Carbs – so use a low a low calorie, low protein, high fat Keto diet.

And then you launch waves “Pulses” of other attacks: Glucose inhibitors and or glutamine inhibitors – the two big food sources for cancer. Your normal body cells uses glucose and glutamine, so you don’t live like this all the time; but for a short attack your normal body cells can cope… but the cancer cells cannot. The idea of it also being a low protein diet is that cancers can also use proteins as a food source – so you limit this route as well – they can’t do much to extract energy from fat so that is high in the diet.

Pulse Press on an individual:

Matching this  “Press Pulse” together with the above: Ivermectin interferes with cancer cells in many ways, one of which is as a glutamine inhibitor, Fenbendazole is also, amongst other things, a glucose inhibitor. Though I don’t think Dr.Thomas Seyfried uses either, In the video he says he uses low dose 6-diazo-5-oxo-L-norleucine (DON) as a glutamine inhibitor. The low does is effective and has far less toxicity than higher doses, though there are ways being looked at to mitigate the toxicity at higher doses while at the same time making it a lot more potent against the cancer : https://pmc.ncbi.nlm.nih.gov/articles/PMC10049321/  . The Other Pulse stresses are he uses are Chemo and also HBOT and Vitamin C IV Drips. Maybe other IV drips like Dr.Tullio Simoncini’s Sodium Bicarbonate can be used as well https://youtu.be/ixF0Lf9Gtv4 . https://simoncinicancertherapy.com/treatment-protocols/ . https://x.com/i/status/1884456191830471115 . This patient uses a heaped table spoon of Sodium Bicarb:

As with lots of these things, dosage & Scheduling vary with different proponents of the idea:  https://elsagamma.decorexpro.com/en/lechenie-sodoj-po-neumyvakinu-kak-prinimat/

but https://www.biblik.sk/news/dr-i-p-neumyvakin-healing-with-soda-as-a-prevention-and-using-soda-to-cure-diseases-and-return-health/ 

https://www.youtube.com/@seoleoint . .    https://x.com/i/status/1763795851934044452 https://youtu.be/hrUupHg0wQ0  Thinking aloud, it might be an idea to mix 2DG – see below – with Sodium Bicarbonate 

Sample Simoncini protocols:

Stomach cancer: “360° TAT (turn around treatment)
When sodium bicarbonate administered in a cavity
Lay down on the bed
2 pillows under the pelvis
Turn around 90° degrees every 15 minutes
Positions: supine, left and right side, prone
Stomach cancer

1 plenty teaspoon sodium bicarbonate in 1 and 1/2 glass tepid (60 degrees) water
Taste with a finger if it’s salty
Lay down on the bed
Drink slowly and make TAT
Twice a day, 20 minutes before breakfast and dinner, for one month
1 week break
Make 2 times the whole cycle

Peritoneum cancer: IV 500 ml sodium bicarbonate 5% 6 days on 6 days off for 4 cycles. Locate a trans abdominal intra-peritoneal catheter. Administer 300-400-500 ml 5% sodium bicarbonate every day for 4-5 days, then every other day for 2 weeks. Take 5 days break then repeat the whole cycle. Then Cat Scan. When in the day break, put 10-20 ml in the catheter”

Ivermectin meets Press Pulse as it blocks Glucose & Glutamine. Interesting aside in this video that NAC switches off the power of Ivermectin.

 Dr.Tullio Simoncini is one of those “We’ve run him out of doctors” but even oral alkalising therapy shows amazing results “The median overall survival from the start of alkalization therapy of the patients with high urine pH (>7.0) was significantly longer than those with low urine pH (≤ 7.0) 16.1 months compared to 4.7 months” https://pubmed.ncbi.nlm.nih.gov/32014931/ 

“The mean urine pH of the Sodium bicarbonate & IV Vitamin C  group was significantly higher than that of the control group (7.32±0.45 vs. 6.44±0.74, respectively; p<0.005). The median overall survival for the intervention group was 44.2 months, as compared with 17.7 months for the control group. 3g to 5g of Sodium Bicarbonate by mouth per day and 25g to 50 g of IV vitamin C each day (a low dose for IV vitamin C – so maybe results could have been better ” https://europepmc.org/article/med/35399313

“Cells enter a state of dormancy as tissues starved of oxygen become increasingly acidic, his dormancy, thought to be a major cause of drug resistance and disease relapses in cancer, might be relatively easy to reverse when it is induced by acidity acidic conditions halt protein synthesis in cells.  When a cell becomes acidic, lysosomes—sac-like organelles that break down proteins and recycle their amino acids—rapidly disperse from their location near the nucleus to the periphery in response to acidity, cells turn off a critical molecular switch called mTORC. In ordinary conditions mTORC gauges the availability of nutrients before giving cells the green light to grow and divide. The silencing of mTOR shuts down production of proteins, disrupting the cells’ metabolic activity and circadian clocks, pushing them into quiescence. Tumors taken from mice that had been given baking soda in their drinking water light up with mTOR activity. It is possible that if baking soda can be used to reawaken such dormant cells, tumors might become far more sensitive to therapy. In acidic conditions, lysosomes rapidly disperse carrying mTOR away to the cell’s periphery and separating it from a protein called RHEB found near the nucleus and required for mTOR activation. Lacking one of its key activation signals, mTOR remains dormant, suspending the synthesis of proteins. The team observed that baking soda can reverse this effect, sending lysosomes back to the nucleus where RHEB waits, restoring mTOR activity. https://www.pennmedicine.org/news/news-releases/2018/june/how-might-baking-soda-boost-cancer-therapy 

Sodium Bicarbonate plus chemotherapy ? Sometimes yes, sometimes no: “Surprisingly, extracellular alkalization induced a 2- to 3-fold increase in the efficacy of doxorubicin.²⁹ However, while sodium bicarbonate increases the uptake of weak-base drugs through elevating the pHe, it greatly reduces the efficacy of some weak acidic chemotherapeutics, such as chlorambucil.^37,38 Thus, it is not wise to combine baking soda with acidic agents.” – So it’s a good idea to google the question: “Is X Chemo agent acid or base” ? https://pmc.ncbi.nlm.nih.gov/articles/PMC7249593/ 

“we include the experience of a 79-year-old man with widely metastatic renal cancer at the Moffitt Cancer Center. After failing first-line treatment, he discontinued conventional therapy and began a self-administered course of vitamins, supplements, and 60 g of bicarbonate mixed in water daily. As of this submission, he has remained well with stable tumor for 10 months.” https://pmc.ncbi.nlm.nih.gov/articles/PMC7249593/ 

Oral administration of Sodium Bicarbonate allows the NK – Natural Killer – Cells to wake up and attack cancer:   https://pubmed.ncbi.nlm.nih.gov/28195314/ 

You can buy PH testing strips from Amazon and your local chemist to test the PH of your urine and add sodium bicarbonate to your drinks in between meals (so it does not interfere with your digestion and stomach acid too much) to get your PH higher (the higher, the more alkaline and the less acidic. 7 = neutral”) . These studies split their reporting mostly between above and below 7, but for deeply therapeutic effects 7.5 or even 8 is the number to aim for.  Slow release sodium bicarbonate tablets might be a good idea, but I can’t get them yet in the UK: https://pmc.ncbi.nlm.nih.gov/articles/PMC6346694/ . There are few drugs – if any – that get cancer patients to live 44 months rather that 17… and sodium bicarbonate costs literally pennies.

There is a folk recipe called “Trojan Horse” where sodium bi-carbonate is mixed with molasses or maple syrup. The idea is that the sugar in the molasses is gobbled up by the cancer and at the same time it takes in the sodium bicarbonate and it is the sodium bicarbonate that then kills the cancer: https://www.myjourneytoacure.com/blog/baking-soda-and-maple-syrup-history-benefits-and-how-to-use 

Cesium therapy: “Mass spectrographic and isotope studies have shown that potassium, rubidium, and especially cesium are most efficiently taken up by cancer cells. This uptake was enhanced by Vitamins A and C as well as salts of zinc and selenium. The quantity of cesium taken up was sufficient to raise the cell to the 8 pH range. Where cell mitosis ceases and the life of the cell is short. Tests on mice fed cesium and rubidium showed marked shrinkage in the tumor masses within 2 weeks. In addition, the mice showed none of the side effects of cancer. Tests have been carried out on over 30 humans. In each case the tumor masses disappeared. Also all pains and effects associated with cancer disappeared within 12 to 36 hr;” https://pubmed.ncbi.nlm.nih.gov/6522424/

“Self-treatment of cancer with cesium chloride, despite proven lack of efficacy, continues to produce serious adverse effects. Among these is hypokalemia predisposing to life-threatening arrhythmia.” https://academic.oup.com/ckj/article/8/3/335/404822 

Cesium chloride Protocol : https://www.royalrife.com/cesium.html 

Berberine, Curcumin & Milk Thistle block Glutamine Uptake (cancer uses Glutamine  as a food):

Berberine dose depended effects: https://pmc.ncbi.nlm.nih.gov/articles/PMC6580644/ 

It is well known that Black pepper increases the availability and absorption of Curcumin by 20 times, so much so that most curcumin is often sold “with added black pepper” : https://www.hopkinsmedicine.org/health/wellness-and-prevention/turmeric-benefits#:~:text=Combining%20the%20spice%20with%20black,to%20increase%20bioavailability%20by%202000%25. , but you can get another huge boost in this absorption by the use of fenugreek: “Curcumin, combined with fenugreek soluble fibre (galactomannans), has been shown to enhance curcumin bioavailability by increasing the absorption and saturation to up to 20 times compared to curcumin alone” https://pmc.ncbi.nlm.nih.gov/articles/PMC7739318/#:~:text=Curcumin%2C%20combined%20with%20fenugreek%20soluble,alone%20%5B8%2C9%5D.

Curcumin is difficult to absorb so they change the molecule a bit and came up with Theracurmin® – which you can buy as a supplement. “Theracurmin efficiently inhibits the growth of human prostate and bladder cancer cells via induction of apoptotic cell death and cell cycle arrest. ”  https://pubmed.ncbi.nlm.nih.gov/26718024/  

Biocurcumax is another formula  Curcumin, where they redissolve Curcumin in the essential oil of turmeric that has high absorbability https://pubmed.ncbi.nlm.nih.gov/20046768/ 

(It is this Biocurcumax  that is used in combination with with sodium dichloroacetate: Dichloroacetate Treatment with Curcumin  https://ar.iiarjournals.org/content/38/11/6253 “)

This sodium dichloroacetate therapy dosage calculator https://www.dcaguide.org/dca-information/dca-dosage-and-usage-long-guide/ can then be used as a base to use the lowest dose of sodium dichloroacetate and hen reduce it a bit further and then add the Biocurcumax to get a “dramatically enhanced inhibition of cell growth” without the side effects. 

Mixing curcumin with Lecithin also increases absorbability of other anti-cancer things like:    https://onlinelibrary.wiley.com/doi/full/10.1002/app.56816 

Using oil, Lecithin &  fenugreek will possibly help with the absorption of the likes of https://pmc.ncbi.nlm.nih.gov/articles/PMC10011078/

Astaxanthin:  Recently, many in vivo and in vitro studies have confirmed that astaxanthin inhibits the growth of various tumor cells, such as neuroblastoma, lung cancer, gastric cancer, oral cancer, colon cancer, breast cancer, bladder cancer, liver cancer and leukemia https://pmc.ncbi.nlm.nih.gov/articles/PMC7459748/#:~:text=Recently%2C%20many%20in%20vivo%20and,24%2C25%2C26%5D. 

Black currant extract: “Anthocyanin-rich black currant extract suppresses the growth of human hepatocellular carcinoma cells” : https://pubmed.ncbi.nlm.nih.gov/21121259/ 

“blackcurrant extract inhibits proliferation of the MCF10A healthy human breast epithelial cell line through induction of G0/G1 arrest and apoptosis” https://www.spandidos-publications.com/10.3892/mmr.2017.7391 

Matcha green tea (MGT): “we found that the natural compound Matcha green tea mechanistically targets oxidative phosphorylation and therefore CSC propagation. Importantly, we demonstrated that MGT effectively down-regulated oxygen consumption rate (OCR) and extracellular acidification rate (ECAR). Moreover, MGT treatment impaired others cellular metabolic pathways, and several cell signaling pathways such as cell cycle regulation, antioxidant response and inflammation. ” https://pmc.ncbi.nlm.nih.gov/articles/PMC6128439/ 

Pumpkin seeds “extract of hull-less pumpkin seed (HLPS) has a significant anti-cancer effect.” https://pmc.ncbi.nlm.nih.gov/articles/PMC8017459/

Ozonated water: – You bubble Ozone (use an Ozone generating machine) through a glass of water. https://pmc.ncbi.nlm.nih.gov/articles/PMC4632793/

“Local administration of ozonated water (208 mM) was not associated with any detrimental effects in normal tissues. On the other hand, local administration of ozonated water (20.8, 41.6, 104, or 208 mM) directly into the tumor tissue induced necrosis and inhibited proliferation of tumor cells. There was no significant difference in the number of terminal deoxynucleotidyl transferase-mediated deoxyuridine triphosphate-biotin nick-end labelling (TUNEL)-positive cells following administration of ozonated water. The size of the necrotic areas was dependent on the concentration of ozonated water. These results indicate that ozonated water does not affect normal tissue and damages only the tumor tissue by selectively inducing necrosis. There is a possibility that it exerts through the production of reaction oxygen species (ROS). In addition, the induction of necrosis rather than apoptosis is very useful in tumor immunity. “

“A single Tigilanol Tiglate treatment resulted in 75% complete response ” https://pubmed.ncbi.nlm.nih.gov/32542733/ . “Tigilanol Tiglate EBC-46 is an extract of  seeds of the pink fruit of the blushwood tree, Fontainea picrosperma. Marsupials such as musky rat-kangaroos that eat blushwood fruit avoid the tigilanol tiglate-rich seeds, which when ingested trigger vomiting and diarrhea. Injecting far smaller doses of EBC-46 directly into some solid tumors modifies the cellular signaling by PKC. Specifically, EBC-46 is proposed to activate certain forms of PKC, which in turn influence the activity of various proteins in the cancerous cells, attracting an immune response by the host’s body. The resulting inflammation makes the tumor’s vasculature, or blood vessels, leaky, and this hemorrhaging causes the tumorous growth to die. In the case of external, cutaneous malignancies, the tumors scab up and fall off, and ways of delivering EBC-46 to internal tumors are being investigated.” https://news.stanford.edu/stories/2022/10/breakthrough-production-acclaimed-cancer-treating-drug

EGCG (Epigallocatechin Gallate) – an extract of Green Tea: https://www.youtube.com/watch?v=Em1qbnUEGq8

EGCG regulated nasopharyngeal carcinoma Cancer stem cells, their self-renewal capacity, and inhibited their invasive characteristics. It supports the pivotal role of EGCG as a dietary compound targeting nasopharyngeal carcinoma Cancer and may decrease recurrence and metastasis in nasopharyngeal carcinoma cells. https://pmc.ncbi.nlm.nih.gov/articles/PMC3575296/ 

This pattern of the pulsing 3 days on, 3 days off is seen working in CBD oil and looks like it might be a standard pattern to use with more cancer attacking agents. It looks like you beat the cancer up for a few days, then back off and it re-builds a bit, then you hit it again and repeat, each time diminishing the cancer. While if you attack it continuously, it will change and put up more defences to avoid this attack altogether in future. 

Anti-parasitic Herbs: All of these have powerful anti-cancer properties as well:

Green Walnut Hull (Black Walnut, but early in the season) :  “The results showed that the green walnut husk extracts had an anti-gastric cancer effect mainly by inhibiting the invasion of gastric cancer cells and inducing its apoptosis.” https://pmc.ncbi.nlm.nih.gov/articles/PMC8899360/#:~:text=Rna%2Dseq%20detected%20the%20expression,cells%20and%20inducing%20its%20apoptosis.

Black Walnut extracts: “The results showed that ellagic acid, juglone, and the “strong acids” fraction depressed the tumor growth rate significantly.”   https://www.sciencedirect.com/science/article/abs/pii/S002235491536528X  

Walnut Leaves: “The results showed that walnut leaves possess enormous potential as antioxidants, and as anticancer and antifungal agents.”  https://www.mdpi.com/2073-4395/13/5/1232 

Sweet Wormwood (Artemisia annua) : https://pmc.ncbi.nlm.nih.gov/articles/PMC2629082/  “Taken together, our results demonstrate that a key event in the artemisinin anti-proliferative effects in prostate cancer cells is the transcriptional down-regulation of CDK4 expression by disruption of Sp1 interactions with the CDK4 promoter.” – unpublished work shows that this works best on a 3 days on, 3 days off routine.

“20 years ago, back in 2005, it was found that when they chemically tagged wormwood with iron, actually transferrin, this was super absorbed into the cancer cells like a Trojan horse as the cancer cells like iron, this combination then blew-up inside the cancer cell and killed it” https://pubmed.ncbi.nlm.nih.gov/16185154/ . Development on this work looks like it has been squashed as nothing more seems to have happened in 20 years.

Cloves: https://pmc.ncbi.nlm.nih.gov/articles/PMC4132639/ : “Clove Extract Inhibits Tumour Growth and Promotes Cell Cycle Arrest and Apoptosis”

Black Walnut: Juglone (From Black Walnuts hulls) – induced apoptosis in human gastric cancer SGC-7901 cells via the mitochondrial pathway: https://pubmed.ncbi.nlm.nih.gov/19815401/ 

Chinese herbs: https://pmc.ncbi.nlm.nih.gov/articles/PMC6142080/  

Soursop: “The soursop leaves endophytic fungi extracts showed cytotoxicity against cervical cancer cells by inhibiting the multiplication of HeLa cancer cells in vitro” https://pmc.ncbi.nlm.nih.gov/articles/PMC9256652/#b0110  

WZ35 a synthetic – more soluble – form of curcumin  inhibits gastric cancer cell metastasis by depleting glutathione to promote cellular metabolic remodelling – not available to public – but mixing with warm olive oil – it’s fat soluble – and black pepper in crease it’s bioavailability. “Curcumin is one of those amazing things that blocks loads of things”. Betulinic acid, which is found in white birch bark, and also Neem bark to a lesser extent, is very good at blocking Glutamine: 

Turkey Tail Mushrooms: https://pmc.ncbi.nlm.nih.gov/articles/PMC6889544  “Trametes versicolor (Tv), commonly known as Turkey tail is known to enhance innate and adaptive immune responses

Skull Cap Mushrooms https://www.mdpi.com/1424-8247/16/2/302#:~:text=Scutellaria%20root%20contains%20the%20flavonoid,125%2C126%2C127%5D. “Autophagy Induction by Scutellaria (Skull cap) Flavones in Cancer: Recent Advances

What is a “scream out” in effective cancer treatment, is combination treatments. These don’t lend themselves to big Phase 3 trials as the number of combinations and permutations (includes order) of just as handful of treatments quickly becomes huge.

Here you see Ivermectin matched with the over the counter lactoferrin working wonders:

This mix looks like it is getting the same sort of amazing results as the B-17 & pancreatic enzymes combo. This one of the best Ivermectin videos I’ve seen:  Huge tumours shrinking in weeks. The thinking being that the lactoferrin breaks down the protective outside of the cancer cells allowing the Ivermectin to get in and mess up the cancer cell. They spread their Ivermectin out over the day – so spread the dose over 3 to 4 times a day eating food at the same time: https://rumble.com/v21o4vs-dr-chetty-and-dr-landrito-ivermectin-and-lactoferrin-for-cancer.html .They start off with low doses and build up to higher doses 1.5 g per KG, 2 g per day or even higher, being careful to keep under the side-effect level and lowering the dose if symptoms occur .

Halo-Lactoferrin is loaded with iron and works well for triple negative breast cancer: https://pmc.ncbi.nlm.nih.gov/articles/PMC7847686/ , while Apo-lactoferrin (the standard one you buy in health food shops etc) is usually better for most other cancers: “Anticancer effects of lactoferrin: underlying mechanisms” :  https://academic.oup.com/nutritionreviews/article-abstract/72/12/763/1840536

Lactoferrin sweeps up free iron in the blood, this is then absorbed in the intestines if needed and used to make fresh iron rich blood cells, otherwise the iron is excreted away. Cancer cells, and other pathogens use this free iron and so are weakened when they don’t have access to it. Despite instructions on the packaging, Lactoferrin is best taken on an empty stomach so at least 15 mins before a meal, so it can be absorbed before the meal causes the acidity of the stomach to increase.

These doctors also use B17 and sodium bicarbonate (up to 2 teaspoons a day) . One doctor uses a catheter to wash the perineum with sodium bi-carb. So just to reemphasise this is a a super video that is well worth a watch:  https://rumble.com/v21o4vs-dr-chetty-and-dr-landrito-ivermectin-and-lactoferrin-for-cancer.html .

Another combination treatment that is getting results is Dichloroacetate Treatment with Curcumin  https://ar.iiarjournals.org/content/38/11/6253  

“Long-term stabilization of stage 4 colon cancer using sodium dichloroacetate therapy” https://pmc.ncbi.nlm.nih.gov/articles/PMC5067498/ 

The Abscopal Effect – you attack tumour with a proton beam, or inject it with a flu shot and this triggers the immune system to clear all the tumours: https://www.cancer.gov/news-events/cancer-currents-blog/2020/cancer-abscopal-effect-radiation-immunotherapy#:~:text=The%20abscopal%20effect%20occurs%20when,tumors%20elsewhere%20in%20the%20body.

“Sodium Butyrate Selectively Kills Cancer Cells and Inhibits Migration in Colorectal Cancer by Targeting Thioredoxin-1 ” https://pmc.ncbi.nlm.nih.gov/articles/PMC7263851/ 

MR1: Back in 2020 Cardiff University fixed cancer – all cancers – using a T-Cell like this: “Here, we use genome-wide CRISPR–Cas9 screening to establish that a T cell receptor (TCR) recognized and killed most human cancer types via the monomorphic MHC class I-related protein, MR1, while remaining inert to noncancerous cells. Unlike mucosal-associated invariant T cells, recognition of target cells by the TCR was independent of bacterial loading. Furthermore, concentration-dependent addition of vitamin B-related metabolite ligands of MR1 reduced TCR recognition of cancer cells, suggesting that recognition occurred via sensing of the cancer metabolome (metabolome: definition: “the qualitative and quantitative collection of all low molecular weight molecules (metabolites) present in a cell that are participants in general metabolic reactions and that are required for the maintenance, growth and normal function of a cell”) . An MR1-restricted T cell clone mediated in vivo regression of leukemia and conferred enhanced survival of NSG mice. TCR transfer to T cells of patients enabled killing of autologous and nonautologous melanoma. These findings offer opportunities for HLA-independent, pan-cancer, pan-population immunotherapies.”  https://www.nature.com/articles/s41590-019-0578-8 – The Journal of International immunology published this in 2021 https://academic.oup.com/intimm/article/34/3/141/6412733 , other than that, it has been sat on ever since – word on the street is it’s been knobbled – how many more are out there like this ?

“IP6 possesses other significant benefits for human health, such as the ability to enhance immune system, prevent pathological calcification and kidney stone formation, lower elevated serum cholesterol, and reduce pathological platelet activity. In this review we show the efficacy and discuss some of the molecular mechanisms that govern the action of this dietary agent. Exogenously administered IP(6) is rapidly taken up into cells and dephosphorylated to lower inositol phosphates, which further affect signal transduction pathways resulting in cell cycle arrest. A striking anticancer action of IP(6) was demonstrated in different experimental models. In addition to reducing cell proliferation, IP(6) also induces differentiation of malignant cells. Enhanced immunity and antioxidant properties also contribute to tumor cell destruction. Preliminary studies in humans show that IP6 and inositol, the precursor molecule of IP6, appear to enhance the anticancer effect of conventional chemotherapy, control cancer metastases, and improve quality of life.”  https://pubmed.ncbi.nlm.nih.gov/17044765/ 

IP6 Video

IP-6 treated mice on top row  – 2 weeks:  

IP6 plus inositol induced complete remission in stage IV melanoma: https://pubmed.ncbi.nlm.nih.gov/30615010/  

“The most consistent and best anticancer results were obtained from the combination of IP₆ plus inositol. In addition to reducing cell proliferation, IP₆ increases differentiation of malignant cells, often resulting in a reversion to normal phenotype. Exogenously administered IP6 is rapidly taken into the cells and dephosphorylated to lower-phosphate inositol phosphates, which further interfere with signal transduction pathways and cell cycle arrest. Enhanced immunity and antioxidant properties can also contribute to tumor cell destruction.” https://www.sciencedirect.com/science/article/pii/S002231662302535X#:~:text=Exogenously%20administered%20IP6%20is,contribute%20to%20tumor%20cell%20destruction. 

I use IP6 Gold:  https://www.amazon.co.uk/inositol-Gold-Powder-308g-unflavoured/dp/B08V5NRQ21

Colloidal also called Nano Silver : https://www.sciencedirect.com/science/article/pii/S0753332222009003

Fucoidan: “Fucoidan, a natural component of brown seaweed, has anti-cancer activity against various cancer types by targeting key apoptotic molecules. It also has beneficial effects as it can protect against toxicity associated with chemotherapeutic agents and radiation. Thus the synergistic effect of fucoidan with current anti-cancer agents is of considerable interest.”  https://pmc.ncbi.nlm.nih.gov/articles/PMC4413214/ 

2 deoxy d glucose “2DG” “2DG is a non-metabolising glucose analog – a fake glucose, that targets glucose metabolism to deplete cancer cells of energy. In addition, 2DG increases oxidative stress, inhibits N-linked glycosylation, and induces autophagy. It can efficiently slow cell growth and potently facilitate apoptosis in specific cancer cells. Although 2DG itself has limited therapeutic effect in many types of cancers, it may be combined with other therapeutic agents or radiotherapy to exhibit a synergistic anticancer effect… but not for me as a side effect can be… “Stomach pain or aggravation of pre-existing problems in those with unhealed gastrointestinal ulcers or erosions”  : https://pubmed.ncbi.nlm.nih.gov/25218591/

https://www.2dg.org 

https://cellandbioscience.biomedcentral.com/articles/10.1186/s13578-023-01137-w

Electricity: https://pmc.ncbi.nlm.nih.gov/articles/PMC8788921/#:~:text=Electrical%20stimulation%20could%20modulate%20the,with%20these%20immune%20agents%20externally.  “The study showed immune system activation after treatment, and that electrical pulses increase the effectiveness of the chemotherapeutic drugs by permeabilizing tumor cells and creating “holes” or openings for the drugs to enter. The drug used for these experiments was bleomycin (BLM). 5 mg/kg (100 µg per mouse) of bleomycin was injected intravenously into the A/J mice and electrical pulses (8 square wave pulses of 1.04 kV amplitude, 100 us pulse width and 1 Hz frequency) were additionally delivered to some of the mice by two flat and parallel stainless-steel electrodes placed 8 mm apart at the opposite margins of the fibrosarcoma SA-1 tumor. Treatment with BLM or electrical pulses alone induced moderate antitumor effect. However, treatment was significantly more effective with combined BLM and ECT. It was found that 52% of the tumors responded completely in 120 days, and were determined cured. The remaining 10 tumors regrew with some delay after approximately 10 days in partial response. Increased immune resistance was evaluated by determining the phagocytic activity and the ability to elicit oxidative bursts by monocytes and polymorphonuclear granulocytes after ECT. The percentage of monocytes that were able to elicit oxidative burst was increased 7 days after the ECT treatment but then returned to normal after 14 days. Immune responses were measured by blast transformation tests of spleen mononuclear cells after electrochemotherapy, and it was found that T lymphocyte activity increased 14 days after ECT. White blood cell count and the number of monocytes were also increased. These generated T cells could potentially have the ability to kill non-ECT-sensitive cancer cells within the primary tumour, limit metastatic spreading, and be responsible for the absence of local relapses (Kroemer et al., 2013) as shown in Figure 3.”

Electricity: Cure rate of “50%–63% in the treatment of Basal Cell Carcinoma (BCC)”  https://www.sciencedirect.com/science/article/pii/S2590137022001078

Blue-Green Algae AFA Klamath: https://pubmed.ncbi.nlm.nih.gov/17887936/  – Natural Killer Cell boost: “Ingestion of 1.5 g of dried whole A. flos-aquae resulted in a transient reduction in peripheral blood NK cells in 21 healthy human volunteers, suggesting increased NK cell homing into tissue. We have now identified an extract from A. flos-aquae (AFAe) that directly activates NK cells in vitro and modulates the chemokine receptor profile. NK cell activation was evaluated by expression of CD25 and CD69 on CD3-CD56+ cells after 18 hours. Changes in CXCR3 and CXCR4 chemokine receptor expression after 5-60 minutes were evaluated by immunostaining and flow cytometry. AFAe induced the expression of CD69 on CD3-CD56+ NK cells, induced CD25 expression on 25% of these cells, and acted in synergy with interleukin”

When cancer cells change their food source, Yarrow can help block that: https://pmc.ncbi.nlm.nih.gov/articles/PMC6435158/

Chemotherapy… time of day “relevance of time-of-day of immunochemotherapy for progression-free and overall survival in patients with non-small cell lung cancer”: https://ascopubs.org/doi/10.1200/JCO.2025.43.16_suppl.8516 

Cannabis oil:

Cannabis oil use of suppositories – rectal administration

Methylene Blue

Published Science on Methylene Blue:

https://pubmed.ncbi.nlm.nih.gov/38503388/ 

“Results: After intravenous administration of Methylene Blue at 10-20 mg/kg, it quickly transitioned in the tumour to a colourless leucomethylene blue, with maximum accumulation in the tumour occurring after 5-10 min. A concentration of 10 mg/kg resulted in a relative increase of the tumour oxygenation level for small tumours (volume 50-75 mm³) and normal tissue 120 min after the introduction of MB. A shift in tumour metabolism towards oxidative phosphorylation (according to the lifetime of the NADH coenzyme) was measured using FLIM method after intravenous administration of 10 mg/kg of MB. Intravenous administration of MB at 20 mg/kg results in a long-term decrease in oxygenation, which persisted for at least 120 min after the administration and did not return to its initial level. Conclusions: Administration of MB at 10 mg/kg shown to increase tumour oxygenation level, potentially leading to more effective antitumor therapy. However, at higher doses (20 mg/kg), MB may cause long-term decrease in oxygenation.”

Intravenous Vitamin C : https://www.science.org/doi/10.1126/scitranslmed.3007154 “Because of marked pharmacokinetic differences, intravenous, but not oral, ascorbate produces millimolar concentrations both in blood and in tissues, killing cancer cells without harming normal tissues. In the interstitial fluid surrounding tumor cells, millimolar concentrations of ascorbate exert local pro-oxidant effects by mediating hydrogen peroxide (H₂O₂) formation, which kills cancer cells. We investigated downstream mechanisms of ascorbate-induced cell death. Data show that millimolar ascorbate, acting as a pro-oxidant, induced DNA damage and depleted cellular adenosine triphosphate (ATP), activated the ataxia telangiectasia mutated (ATM)/adenosine monophosphate–activated protein kinase (AMPK) pathway, and resulted in mammalian target of rapamycin (mTOR) inhibition and death in ovarian cancer cells. The combination of parenteral ascorbate with the conventional chemotherapeutic agents carboplatin and paclitaxel synergistically inhibited ovarian cancer in mouse models and reduced chemotherapy-associated toxicity in patients with ovarian cancer.”

Again, the line “from the BBC” types is “there is not enough evidence to show…” they’ve had 40, 50 years to get this evidence and they have not done so – they’ve no intention of ever doing it and will actively work to prevent others doing it. They clearly have a policy of “Do not cross line” with this sort of thing so: “there is not enough evidence… and we’ll dam well make sure it stays like that” is not a valid scientific argument, it is a Mafia policy statement that has been issued from a lawyers office on Wall St to protect the interests of their clients on the stock market . 

So these therapies are safe Immunological treatments: IMM-101, Low dose Naltrexone, Bromelain, Turkey Tail Mushrooms that get your immune system boosted so it can kill the cancer and safe Chemo treatments… : Ivermectin, Fenbendazole, Methylene Blue.. to chemically weaken the cancer and kill the cancer as well. Weakening the a low calorie Keto diets is also in there. These should be available to available to all cancer patients, they are being blocked. This is unacceptable.

At the start of this article I showed what can be done, but what is not done as standard, for high end bad backs. How did I fix that back ? The videos of the treatment are here: https://goodback.co.uk/2024/12/25/back-pain-videos-for-the-better-than-surgery-asmi-advanced-spinal-mobilisation-machine/ along with many others. In the current parlance of this post I used a “safe Immunological treatment”: I got his white blood cells to fix him. The results were dramatic and fast. Which is quite standard with this approach. All it was was a bit of Ultrasound to relieve the deep muscle spasms he had that were blocking lymph drainage and so were blocking the white blood cells getting in and doing the repair needed and then a few minutes of EMS – Electo-muscular Stimulation – to pump out the old stale lymph. Repeat a few times and job done ! His immune system did the repair work and he was back on his feet in no time, pain free and back to a full life – this after the world’s top back doc the “Back Mechanic” himself, Dr Stuart McGill  had told him to go for surgery as his back was “beyond even me” – that’s a great visible for how powerful the immune system is in bad backs – its the same in cancer.

This is a list produced by AI that ranks the best repurposed drugs and herbs for cancer: https://www.onedaymd.com/2025/01/ranking-top-19-terminal-cancer.html 

Paricalcitol, hydroxychloroquine, intravenous vitamin C, statins, metformin, curcumin, and aspirin: “Theracurmin®, is currently available and appears to provide significantly greater blood levels and greater hopes of efficacy than Curcumin.”  https://pmc.ncbi.nlm.nih.gov/articles/PMC6049054/   

“Ivermectin Cocktail Blocks Metastases in Key Cancers (2025)” : https://www.onedaymd.com/2025/03/ivermectin-cocktail-blocks-metastases.html 

Some case studies:  https://www.onedaymd.com/2024/12/surviving-colon-cancer-with-ivermectin.html

Cancer Stem killing drug: Cymirafen : https://www.youtube.com/watch?v=FXyYX–NI0k

Hydrogen Peroxide: Bill Munro. “For every inhale I pump the 3 percent Hydrogen peroxide 5 or 6 times and do 6 or 7 of these a day”

Deuterium depleted water https://pubmed.ncbi.nlm.nih.gov/31519768/ 

Oral recombinant methioninase  https://ar.iiarjournals.org/content/40/5/2813

Fibre, time of day, time of therapy…. https://www.youtube.com/watch?v=RUnHF6EDHtk

Sources: The internet , Amazon – but fakes are an issue, and then for more difficult to get hold of products:

https://www.reliablehealthcareindia.com  https://www.tulienterprise.com/  AllDayChemist.com  https://www.getwellmedex.com/  https://buyer.indiamart.com/